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Daniela Cota

Showing results (31-40 of 101) with videos related to

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Diabetes|September 9, 2008
Fatty acid synthase inhibitors modulate energy balance via mammalian target of rapamycin complex 1 signaling in the central nervous systemKarine Proulx, Daniela Cota, Stephen C Woods, et al.
Nature Reviews. Endocrinology|January 15, 2024
mTORC1 in energy expenditure: consequences for obesityCamille Allard, Cristina Miralpeix, Antonio J López-Gambero, et al.
Neuropharmacology|June 6, 2017
Endocannabinoid modulation of homeostatic and non-homeostatic feeding circuitsBenjamin K Lau, Daniela Cota, Luigia Cristino, et al.
Endocrine Reviews|November 25, 2005
The emerging role of the endocannabinoid system in endocrine regulation and energy balanceUberto Pagotto, Giovanni Marsicano, Daniela Cota, et al.
Brain Research Reviews|December 21, 2005
Cannabinoids, opioids and eating behavior: the molecular face of hedonism?Daniela Cota, Matthias H Tschöp, Tamas L Horvath, et al.
Scientific Reports|October 5, 2021
Mass spectrometry imaging of mice brain lipid profile changes over time under high fat dietGianluca Sighinolfi, Samantha Clark, Landry Blanc, et al.
Translational Psychiatry|March 16, 2021
Inhibition of mTOR signaling by genetic removal of p70 S6 kinase 1 increases anxiety-like behavior in miceMuriel Koehl, Elodie Ladevèze, Caterina Catania, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 11, 2008
The role of hypothalamic mammalian target of rapamycin complex 1 signaling in diet-induced obesityDaniela Cota, Emily K Matter, Stephen C Woods, et al.
Frontiers in Pharmacology|June 6, 2018
NPV-BSK805, an Antineoplastic Jak2 Inhibitor Effective in Myeloproliferative Disorders, Causes Adiposity in Mice by Interfering With the Action of LeptinMagalie Haissaguerre, Amandine Ferriere, Samantha Clark, et al.
Molecular Metabolism|August 18, 2019
mTORC1 and CB1 receptor signaling regulate excitatory glutamatergic inputs onto the hypothalamic paraventricular nucleus in response to energy availabilityWilfrid Mazier, Nicolas Saucisse, Vincent Simon, et al.
Pageof 11

Showing results (31-40 of 101) with videos related to

Sort By:
Pageof 11
Diabetes|September 9, 2008
Fatty acid synthase inhibitors modulate energy balance via mammalian target of rapamycin complex 1 signaling in the central nervous systemKarine Proulx, Daniela Cota, Stephen C Woods, et al.
Nature Reviews. Endocrinology|January 15, 2024
mTORC1 in energy expenditure: consequences for obesityCamille Allard, Cristina Miralpeix, Antonio J López-Gambero, et al.
Neuropharmacology|June 6, 2017
Endocannabinoid modulation of homeostatic and non-homeostatic feeding circuitsBenjamin K Lau, Daniela Cota, Luigia Cristino, et al.
Endocrine Reviews|November 25, 2005
The emerging role of the endocannabinoid system in endocrine regulation and energy balanceUberto Pagotto, Giovanni Marsicano, Daniela Cota, et al.
Brain Research Reviews|December 21, 2005
Cannabinoids, opioids and eating behavior: the molecular face of hedonism?Daniela Cota, Matthias H Tschöp, Tamas L Horvath, et al.
Scientific Reports|October 5, 2021
Mass spectrometry imaging of mice brain lipid profile changes over time under high fat dietGianluca Sighinolfi, Samantha Clark, Landry Blanc, et al.
Translational Psychiatry|March 16, 2021
Inhibition of mTOR signaling by genetic removal of p70 S6 kinase 1 increases anxiety-like behavior in miceMuriel Koehl, Elodie Ladevèze, Caterina Catania, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 11, 2008
The role of hypothalamic mammalian target of rapamycin complex 1 signaling in diet-induced obesityDaniela Cota, Emily K Matter, Stephen C Woods, et al.
Frontiers in Pharmacology|June 6, 2018
NPV-BSK805, an Antineoplastic Jak2 Inhibitor Effective in Myeloproliferative Disorders, Causes Adiposity in Mice by Interfering With the Action of LeptinMagalie Haissaguerre, Amandine Ferriere, Samantha Clark, et al.
Molecular Metabolism|August 18, 2019
mTORC1 and CB1 receptor signaling regulate excitatory glutamatergic inputs onto the hypothalamic paraventricular nucleus in response to energy availabilityWilfrid Mazier, Nicolas Saucisse, Vincent Simon, et al.
Pageof 11