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Annals of Neurology|March 5, 2014
A novel mechanism for cyclic adenosine monophosphate-mediated memory formation: Role of amyloid betaRoberta Ricciarelli, Daniela Puzzo, Olga Bruno, et al.
Expert Review of Neurotherapeutics|November 12, 2009
MAPK, beta-amyloid and synaptic dysfunction: the role of RAGENicola Origlia, Ottavio Arancio, Luciano Domenici, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 20, 2017
Amyloid-β Peptide Is Needed for cGMP-Induced Long-Term Potentiation and MemoryAgostino Palmeri, Roberta Ricciarelli, Walter Gulisano, et al.
Molecular and Cellular Neurosciences|December 11, 2013
The schizophrenia susceptibility gene DTNBP1 modulates AMPAR synaptic transmission and plasticity in the hippocampus of juvenile DBA/2J miceIan J Orozco, Peter Koppensteiner, Ipe Ninan, et al.
Elife|July 12, 2017
LTP and memory impairment caused by extracellular Aβ and Tau oligomers is APP-dependentDaniela Puzzo, Roberto Piacentini, Mauro Fá, et al.
Scientific Reports|September 2, 2016
Time-dependent reversal of synaptic plasticity induced by physiological concentrations of oligomeric Aβ42: an early index of Alzheimer's diseasePeter Koppensteiner, Fabrizio Trinchese, Mauro Fà, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 26, 2019
Neuromodulatory Action of Picomolar Extracellular Aβ42 Oligomers on Presynaptic and Postsynaptic Mechanisms Underlying Synaptic Function and MemoryWalter Gulisano, Marcello Melone, Cristian Ripoli, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 25, 2010
Danish dementia mice suggest that loss of function and not the amyloid cascade causes synaptic plasticity and memory deficitsRobert Tamayev, Shuji Matsuda, Mauro Fà, et al.
Methods in Molecular Biology (Clifton, N.J.)|June 11, 2018
Preparation of Tau Oligomers After the Protein Extraction from Bacteria and Brain CorticesElentina K Argyrousi, Agnieszka Staniszewski, Russell E Nicholls, et al.
Scientific Reports|September 13, 2014
Notoginsenoside R1 increases neuronal excitability and ameliorates synaptic and memory dysfunction following amyloid elevationShijun Yan, Zhi Li, Hang Li, et al.
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