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Plos One|March 23, 2012
Increased migration of monocytes in essential hypertension is associated with increased transient receptor potential channel canonical type 3 channelsZhigang Zhao, Yinxing Ni, Jing Chen, et al.
American Journal of Hypertension|May 31, 2024
Dietary Cinnamaldehyde Activation of TRPA1 Antagonizes High-Salt-Induced Hypertension Through Restoring Renal Tubular Mitochondrial DysfunctionShiqiang Xiong, Shaoyang Lin, Yingru Hu, et al.
Journal of the American Heart Association|July 17, 2017
Enhanced Mitochondrial Transient Receptor Potential Channel, Canonical Type 3-Mediated Calcium Handling in the Vasculature From Hypertensive RatsBin Wang, Shiqiang Xiong, Shaoyang Lin, et al.
Scientific Reports|May 29, 2015
Mitochondrial respiratory dysfunctions of blood mononuclear cells link with cardiac disturbance in patients with early-stage heart failurePeng Li, Bin Wang, Fang Sun, et al.
Hypertension (Dallas, Tex. : 1979)|November 26, 2008
Increased transient receptor potential canonical type 3 channels in vasculature from hypertensive ratsDaoyan Liu, Dachun Yang, Hongbo He, et al.
Hypertension (Dallas, Tex. : 1979)|January 29, 2019
Caloric Restriction Exacerbates Angiotensin II-Induced Abdominal Aortic Aneurysm in the Absence of p53Peng Gao, Hexuan Zhang, Qin Zhang, et al.
Clinical Science (London, England : 1979)|February 2, 2017
Activation of TRPV4 by dietary apigenin antagonizes renal fibrosis in deoxycorticosterone acetate (DOCA)-salt-induced hypertensionXing Wei, Peng Gao, Yunfei Pu, et al.
Frontiers in Cellular and Infection Microbiology|February 27, 2023
Systematic analysis of lysine crotonylation in human macrophages responding to MRSA infectionHao Zhang, Wei Ma, Haoru Liu, et al.
Heliyon|August 17, 2023
A high-salt diet promotes hypertrophic scarring through TRPC3-mediated mitochondrial Ca2+ homeostasis dysfunctionWeijie Xia, Qianran Wang, Shaoyang Lin, et al.
Cell Reports|August 9, 2018
Deficiency of PKD2L1 (TRPP3) Exacerbates Pathological Cardiac Hypertrophy by Augmenting NCX1-Mediated Mitochondrial Calcium OverloadZongshi Lu, Yuanting Cui, Xing Wei, et al.
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