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Current Neurology and Neuroscience Reports|July 2, 2008
Value of genetic models in understanding the cause and mechanisms of Parkinson's diseaseDarren J Moore, Ted M DawsonThe Journal of Clinical Investigation|August 26, 2011
Parkin reinvents itself to regulate fatty acid metabolism by tagging CD36Nada A Abumrad, Darren J MooreFrontiers in Neuroscience|August 1, 2020
LRRK2 and Protein Aggregation in Parkinson's Disease: Insights From Animal ModelsDylan J Dues, Darren J MooreProgress in Molecular Biology and Translational Science|March 8, 2011
Genetic mouse models of neurodegenerative diseasesAlzbeta Trancikova, David Ramonet, Darren J MooreAntioxidants & Redox Signaling|August 19, 2011
Mitochondrial dysfunction in genetic animal models of Parkinson's diseaseAlzbeta Trancikova, Elpida Tsika, Darren J MooreAdvances in Neurobiology|March 30, 2017
Understanding the GTPase Activity of LRRK2: Regulation, Function, and NeurotoxicityAn Phu Tran Nguyen, Darren J MooreJournal of Parkinson'S Disease|February 20, 2025
Aging, cellular senescence and Parkinson's diseaseYue Ma, Madalynn L Erb, Darren J MooreJournal of Parkinson'S Disease|February 23, 2017
VPS35, the Retromer Complex and Parkinson's DiseaseErin T Williams, Xi Chen, Darren J MooreNeuromolecular Medicine|October 7, 2003
Role for the ubiquitin-proteasome system in Parkinson's disease and other neurodegenerative brain amyloidosesDarren J Moore, Valina L Dawson, Ted M DawsonMolecular Neurodegeneration|August 4, 2023
VPS35 and α-Synuclein fail to interact to modulate neurodegeneration in rodent models of Parkinson's diseaseXi Chen, Elpida Tsika, Nathan Levine, et al.Pageof 9