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Redox Biology
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March 21, 2025
Human lung microvascular endothelial cell protein modification by 2-chlorohexadecanoic acid: RhoA mediates 2-chlorohexadecanoic acid-elicited endothelial activation
Haley L Carlson, David A Ford
Arteriosclerosis, Thrombosis, and Vascular Biology
|
October 26, 2018
MPO (Myeloperoxidase) Caused Endothelial Dysfunction
Celine L Hartman, David A Ford
Frontiers in Physiology
|
May 28, 2020
2-Chlorofatty Aldehyde Elicits Endothelial Cell Activation
Jane McHowat, Shubha Shakya, David A Ford
Frontiers in Physiology
|
October 7, 2022
<i>E. coli</i> strain-dependent lipid alterations in cocultures with endothelial cells and neutrophils modeling sepsis
Kaushalya Amunugama, Daniel P Pike, David A Ford
Journal of Lipid Research
|
March 28, 2015
Identification of glutathione adducts of α-chlorofatty aldehydes produced in activated neutrophils
Mark A Duerr, Rajeev Aurora, David A Ford
Frontiers in Immunology
|
September 7, 2021
Neutrophil Myeloperoxidase Derived Chlorolipid Production During Bacteria Exposure
Kaushalya Amunugama, Grant R Kolar, David A Ford
Journal of Lipid Research
|
June 4, 2021
The lipid biology of sepsis
Kaushalya Amunugama, Daniel P Pike, David A Ford
Antioxidants (Basel, Switzerland)
|
February 25, 2023
Characterization of N-Acetyl Cysteine Adducts with Exogenous and Neutrophil-Derived 2-Chlorofatty Aldehyde
Shubha Shakya, Reagan M McGuffee, David A Ford
Journal of Lipid Research
|
December 26, 2024
Resolving lipoxin A<sub>4</sub>: Endogenous mediator or exogenous anti-inflammatory agent?
Reagan M McGuffee, Matthew A Luetzen, David A Ford
Arteriosclerosis, Thrombosis, and Vascular Biology
|
December 28, 2013
Alpha-chlorofatty acid accumulates in activated monocytes and causes apoptosis through reactive oxygen species production and endoplasmic reticulum stress
Wen-yi Wang, Carolyn J Albert, David A Ford
Page
of 14
Search research articles
Search
Showing results (11-20 of 134) with videos related to
Sort By:
Page
of 14
Redox Biology
|
March 21, 2025
Human lung microvascular endothelial cell protein modification by 2-chlorohexadecanoic acid: RhoA mediates 2-chlorohexadecanoic acid-elicited endothelial activation
Haley L Carlson, David A Ford
Arteriosclerosis, Thrombosis, and Vascular Biology
|
October 26, 2018
MPO (Myeloperoxidase) Caused Endothelial Dysfunction
Celine L Hartman, David A Ford
Frontiers in Physiology
|
May 28, 2020
2-Chlorofatty Aldehyde Elicits Endothelial Cell Activation
Jane McHowat, Shubha Shakya, David A Ford
Frontiers in Physiology
|
October 7, 2022
<i>E. coli</i> strain-dependent lipid alterations in cocultures with endothelial cells and neutrophils modeling sepsis
Kaushalya Amunugama, Daniel P Pike, David A Ford
Journal of Lipid Research
|
March 28, 2015
Identification of glutathione adducts of α-chlorofatty aldehydes produced in activated neutrophils
Mark A Duerr, Rajeev Aurora, David A Ford
Frontiers in Immunology
|
September 7, 2021
Neutrophil Myeloperoxidase Derived Chlorolipid Production During Bacteria Exposure
Kaushalya Amunugama, Grant R Kolar, David A Ford
Journal of Lipid Research
|
June 4, 2021
The lipid biology of sepsis
Kaushalya Amunugama, Daniel P Pike, David A Ford
Antioxidants (Basel, Switzerland)
|
February 25, 2023
Characterization of N-Acetyl Cysteine Adducts with Exogenous and Neutrophil-Derived 2-Chlorofatty Aldehyde
Shubha Shakya, Reagan M McGuffee, David A Ford
Journal of Lipid Research
|
December 26, 2024
Resolving lipoxin A<sub>4</sub>: Endogenous mediator or exogenous anti-inflammatory agent?
Reagan M McGuffee, Matthew A Luetzen, David A Ford
Arteriosclerosis, Thrombosis, and Vascular Biology
|
December 28, 2013
Alpha-chlorofatty acid accumulates in activated monocytes and causes apoptosis through reactive oxygen species production and endoplasmic reticulum stress
Wen-yi Wang, Carolyn J Albert, David A Ford
Page
of 14