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David A Pearce

Showing results (31-40 of 189) with videos related to

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The Journal of Biological Chemistry|October 19, 2014
The human synaptic vesicle protein, SV2A, functions as a galactose transporter in Saccharomyces cerevisiaeMarianna Madeo, Attila D Kovács, David A Pearce
Biochemical and Biophysical Research Communications|March 5, 2003
The yeast model for Batten disease: a role for Btn2p in the trafficking of the Golgi-associated vesicular targeting protein, Yif1pSubrata Chattopadhyay, Paul M Roberts, David A Pearce
Neurology|December 15, 2004
Glutamic acid decarboxylase autoimmunity in Batten disease and other disordersDavid A Pearce, Mark Atkinson, Danilo A Tagle
Journal of Child Neurology|September 10, 2013
Treatment of the Ppt1(-/-) mouse model of infantile neuronal ceroid lipofuscinosis with the N-methyl-D-aspartate (NMDA) receptor antagonist memantineRozzy Finn, Attila D Kovács, David A Pearce
Pediatric Nephrology (Berlin, Germany)|November 13, 2013
Lysosome dysfunction in the pathogenesis of kidney diseasesKameswaran Surendran, Seasson P Vitiello, David A Pearce
Neuroscience Letters|September 17, 2015
Abnormally increased surface expression of AMPA receptors in the cerebellum, cortex and striatum of Cln3(-/-) miceAttila D Kovács, Caitlin Hof, David A Pearce
Proceedings of the National Academy of Sciences of the United States of America|December 9, 2003
A role in vacuolar arginine transport for yeast Btn1p and for human CLN3, the protein defective in Batten diseaseYoojin Kim, Denia Ramirez-Montealegre, David A Pearce
Journal of Neuroscience Research|October 6, 2011
Altered glutamate receptor function in the cerebellum of the Ppt1-/- mouse, a murine model of infantile neuronal ceroid lipofuscinosisRozzy Finn, Attila D Kovács, David A Pearce
Bioscience Reports|November 2, 2017
Lack of specificity of antibodies raised against CLN3, the lysosomal/endosomal transmembrane protein mutated in juvenile Batten diseaseTarah Nelson, David A Pearce, Attila D Kovács
Neurochemistry International|February 15, 2011
Altered sensitivity of cerebellar granule cells to glutamate receptor overactivation in the Cln3(Δex7/8)-knock-in mouse model of juvenile neuronal ceroid lipofuscinosisRozzy Finn, Attila D Kovács, David A Pearce
Pageof 19

Showing results (31-40 of 189) with videos related to

Sort By:
Pageof 19
The Journal of Biological Chemistry|October 19, 2014
The human synaptic vesicle protein, SV2A, functions as a galactose transporter in Saccharomyces cerevisiaeMarianna Madeo, Attila D Kovács, David A Pearce
Biochemical and Biophysical Research Communications|March 5, 2003
The yeast model for Batten disease: a role for Btn2p in the trafficking of the Golgi-associated vesicular targeting protein, Yif1pSubrata Chattopadhyay, Paul M Roberts, David A Pearce
Neurology|December 15, 2004
Glutamic acid decarboxylase autoimmunity in Batten disease and other disordersDavid A Pearce, Mark Atkinson, Danilo A Tagle
Journal of Child Neurology|September 10, 2013
Treatment of the Ppt1(-/-) mouse model of infantile neuronal ceroid lipofuscinosis with the N-methyl-D-aspartate (NMDA) receptor antagonist memantineRozzy Finn, Attila D Kovács, David A Pearce
Pediatric Nephrology (Berlin, Germany)|November 13, 2013
Lysosome dysfunction in the pathogenesis of kidney diseasesKameswaran Surendran, Seasson P Vitiello, David A Pearce
Neuroscience Letters|September 17, 2015
Abnormally increased surface expression of AMPA receptors in the cerebellum, cortex and striatum of Cln3(-/-) miceAttila D Kovács, Caitlin Hof, David A Pearce
Proceedings of the National Academy of Sciences of the United States of America|December 9, 2003
A role in vacuolar arginine transport for yeast Btn1p and for human CLN3, the protein defective in Batten diseaseYoojin Kim, Denia Ramirez-Montealegre, David A Pearce
Journal of Neuroscience Research|October 6, 2011
Altered glutamate receptor function in the cerebellum of the Ppt1-/- mouse, a murine model of infantile neuronal ceroid lipofuscinosisRozzy Finn, Attila D Kovács, David A Pearce
Bioscience Reports|November 2, 2017
Lack of specificity of antibodies raised against CLN3, the lysosomal/endosomal transmembrane protein mutated in juvenile Batten diseaseTarah Nelson, David A Pearce, Attila D Kovács
Neurochemistry International|February 15, 2011
Altered sensitivity of cerebellar granule cells to glutamate receptor overactivation in the Cln3(Δex7/8)-knock-in mouse model of juvenile neuronal ceroid lipofuscinosisRozzy Finn, Attila D Kovács, David A Pearce
Pageof 19