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David A Pearce

Showing results (41-50 of 189) with videos related to

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Metabolic Brain Disease|October 11, 2016
Decreased sensitivity of palmitoyl protein thioesterase 1-deficient neurons to chemical anoxiaMeredith Meyer, Attila D Kovács, David A Pearce
Environmental Microbiome|July 18, 2025
Daily turnover of airborne bacterial communities in the sub-AntarcticLucie A Malard, Peter Convey, David A Pearce
Experimental Cell Research|September 21, 2010
A novel interaction of CLN3 with nonmuscle myosin-IIB and defects in cell motility of Cln3(-/-) cellsAmanda L Getty, Jared W Benedict, David A Pearce
Neurobiology of Disease|February 18, 2006
Selectively increased sensitivity of cerebellar granule cells to AMPA receptor-mediated excitotoxicity in a mouse model of Batten diseaseAttila D Kovács, Jill M Weimer, David A Pearce
Expert Opinion on Medical Diagnostics|March 16, 2013
Diagnosis of neuronal ceroid lipofuscinosis: mutation detection strategiesAmanda L Getty, Paul G Rothberg, David A Pearce
Human Molecular Genetics|March 30, 2013
The role of nonsense-mediated decay in neuronal ceroid lipofuscinosisJake N Miller, Chun-Hung Chan, David A Pearce
Human Molecular Genetics|March 8, 2007
Absence of Btn1p in the yeast model for juvenile Batten disease may cause arginine to become toxic to yeast cellsSeasson Phillips Vitiello, Devin M Wolfe, David A Pearce
Human Molecular Genetics|August 6, 2008
Transcript and in silico analysis of CLN3 in juvenile neuronal ceroid lipofuscinosis and associated mouse modelsChun-Hung Chan, Hannah M Mitchison, David A Pearce
Brain : a Journal of Neurology|June 2, 2006
Another disorder finds its geneDenia Ramirez-Montealegre, Paul G Rothberg, David A Pearce
Human Molecular Genetics|September 11, 2014
The novel Cln1(R151X) mouse model of infantile neuronal ceroid lipofuscinosis (INCL) for testing nonsense suppression therapyJake N Miller, Attila D Kovács, David A Pearce
Pageof 19

Showing results (41-50 of 189) with videos related to

Sort By:
Pageof 19
Metabolic Brain Disease|October 11, 2016
Decreased sensitivity of palmitoyl protein thioesterase 1-deficient neurons to chemical anoxiaMeredith Meyer, Attila D Kovács, David A Pearce
Environmental Microbiome|July 18, 2025
Daily turnover of airborne bacterial communities in the sub-AntarcticLucie A Malard, Peter Convey, David A Pearce
Experimental Cell Research|September 21, 2010
A novel interaction of CLN3 with nonmuscle myosin-IIB and defects in cell motility of Cln3(-/-) cellsAmanda L Getty, Jared W Benedict, David A Pearce
Neurobiology of Disease|February 18, 2006
Selectively increased sensitivity of cerebellar granule cells to AMPA receptor-mediated excitotoxicity in a mouse model of Batten diseaseAttila D Kovács, Jill M Weimer, David A Pearce
Expert Opinion on Medical Diagnostics|March 16, 2013
Diagnosis of neuronal ceroid lipofuscinosis: mutation detection strategiesAmanda L Getty, Paul G Rothberg, David A Pearce
Human Molecular Genetics|March 30, 2013
The role of nonsense-mediated decay in neuronal ceroid lipofuscinosisJake N Miller, Chun-Hung Chan, David A Pearce
Human Molecular Genetics|March 8, 2007
Absence of Btn1p in the yeast model for juvenile Batten disease may cause arginine to become toxic to yeast cellsSeasson Phillips Vitiello, Devin M Wolfe, David A Pearce
Human Molecular Genetics|August 6, 2008
Transcript and in silico analysis of CLN3 in juvenile neuronal ceroid lipofuscinosis and associated mouse modelsChun-Hung Chan, Hannah M Mitchison, David A Pearce
Brain : a Journal of Neurology|June 2, 2006
Another disorder finds its geneDenia Ramirez-Montealegre, Paul G Rothberg, David A Pearce
Human Molecular Genetics|September 11, 2014
The novel Cln1(R151X) mouse model of infantile neuronal ceroid lipofuscinosis (INCL) for testing nonsense suppression therapyJake N Miller, Attila D Kovács, David A Pearce
Pageof 19