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David A Stroud

Showing results (61-70 of 109) with videos related to

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Human Mutation|November 10, 2020
Multiomic analysis elucidates Complex I deficiency caused by a deep intronic variant in NDUFB10Guy Helman, Alison G Compton, Daniella H Hock, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 25, 2022
Sideroflexin 4 is a complex I assembly factor that interacts with the MCIA complex and is required for the assembly of the ND2 moduleThomas D Jackson, Jordan J Crameri, Linden Muellner-Wong, et al.
Science Advances|August 15, 2025
Complex II assembly drives metabolic adaptation to OXPHOS dysfunctionRoopasingam Kugapreethan, Sheik Nadeem Elahee Doomun, Joanna Sacharz, et al.
Mitochondrion|October 16, 2024
An integrated multi-omics approach allowed ultra-rapid diagnosis of a deep intronic pathogenic variant in PDHX and precision treatment in a neonate critically ill with lactic acidosisRodrigo T Starosta, Austin A Larson, Naomi J L Meeks, et al.
Elife|November 5, 2019
Function of hTim8a in complex IV assembly in neuronal cells provides insight into pathomechanism underlying Mohr-Tranebjærg syndromeYilin Kang, Alexander J Anderson, Thomas Daniel Jackson, et al.
Elife|March 19, 2020
Correction: Function of hTim8a in complex IV assembly in neuronal cells provides insight into pathomechanism underlying Mohr-Tranebjærg syndromeYilin Kang, Alexander J Anderson, Thomas Daniel Jackson, et al.
Nature Communications|December 4, 2021
High-intensity training induces non-stoichiometric changes in the mitochondrial proteome of human skeletal muscle without reorganisation of respiratory chain contentCesare Granata, Nikeisha J Caruana, Javier Botella, et al.
Molecular Metabolism|December 28, 2020
Metabolic remodeling of dystrophic skeletal muscle reveals biological roles for dystrophin and utrophin in adaptation and plasticityJustin P Hardee, Karen J B Martins, Paula M Miotto, et al.
The Journal of Experimental Medicine|July 9, 2020
Mutations in the exocyst component EXOC2 cause severe defects in human brain developmentNicole J Van Bergen, Syed Mukhtar Ahmed, Felicity Collins, et al.
Cell|August 6, 2013
Coupling of mitochondrial import and export translocases by receptor-mediated supercomplex formationJian Qiu, Lena-Sophie Wenz, Ralf M Zerbes, et al.
Pageof 11

Showing results (61-70 of 109) with videos related to

Sort By:
Pageof 11
Human Mutation|November 10, 2020
Multiomic analysis elucidates Complex I deficiency caused by a deep intronic variant in NDUFB10Guy Helman, Alison G Compton, Daniella H Hock, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 25, 2022
Sideroflexin 4 is a complex I assembly factor that interacts with the MCIA complex and is required for the assembly of the ND2 moduleThomas D Jackson, Jordan J Crameri, Linden Muellner-Wong, et al.
Science Advances|August 15, 2025
Complex II assembly drives metabolic adaptation to OXPHOS dysfunctionRoopasingam Kugapreethan, Sheik Nadeem Elahee Doomun, Joanna Sacharz, et al.
Mitochondrion|October 16, 2024
An integrated multi-omics approach allowed ultra-rapid diagnosis of a deep intronic pathogenic variant in PDHX and precision treatment in a neonate critically ill with lactic acidosisRodrigo T Starosta, Austin A Larson, Naomi J L Meeks, et al.
Elife|November 5, 2019
Function of hTim8a in complex IV assembly in neuronal cells provides insight into pathomechanism underlying Mohr-Tranebjærg syndromeYilin Kang, Alexander J Anderson, Thomas Daniel Jackson, et al.
Elife|March 19, 2020
Correction: Function of hTim8a in complex IV assembly in neuronal cells provides insight into pathomechanism underlying Mohr-Tranebjærg syndromeYilin Kang, Alexander J Anderson, Thomas Daniel Jackson, et al.
Nature Communications|December 4, 2021
High-intensity training induces non-stoichiometric changes in the mitochondrial proteome of human skeletal muscle without reorganisation of respiratory chain contentCesare Granata, Nikeisha J Caruana, Javier Botella, et al.
Molecular Metabolism|December 28, 2020
Metabolic remodeling of dystrophic skeletal muscle reveals biological roles for dystrophin and utrophin in adaptation and plasticityJustin P Hardee, Karen J B Martins, Paula M Miotto, et al.
The Journal of Experimental Medicine|July 9, 2020
Mutations in the exocyst component EXOC2 cause severe defects in human brain developmentNicole J Van Bergen, Syed Mukhtar Ahmed, Felicity Collins, et al.
Cell|August 6, 2013
Coupling of mitochondrial import and export translocases by receptor-mediated supercomplex formationJian Qiu, Lena-Sophie Wenz, Ralf M Zerbes, et al.
Pageof 11