Search research articles
Contact Us
Filters
Showing results (31-40 of 55) with videos related to
Page
of 6
Sort By:
Journal of Molecular and Cellular Cardiology
|
April 1, 2017
Tropomyosin 1: Multiple roles in the developing heart and in the formation of congenital heart defects
Jennifer England, Javier Granados-Riveron, Luis Polo-Parada, et al.
BMC Genetics
|
June 21, 2013
Low-frequency intermediate penetrance variants in the ROCK1 gene predispose to Tetralogy of Fallot
Julian Palomino Doza, Ana Topf, Jamie Bentham, et al.
Human Molecular Genetics
|
December 19, 2018
Transcriptome alterations in myotonic dystrophy skeletal muscle and heart
Eric T Wang, Daniel Treacy, Katy Eichinger, et al.
Human Molecular Genetics
|
November 2, 2013
High-content screening identifies small molecules that remove nuclear foci, affect MBNL distribution and CELF1 protein levels via a PKC-independent pathway in myotonic dystrophy cell lines
Ami Ketley, Catherine Z Chen, Xin Li, et al.
Genome Medicine
|
August 30, 2020
Systems genetics analysis identifies calcium-signaling defects as novel cause of congenital heart disease
Jose M G Izarzugaza, Sabrina G Ellesøe, Canan Doganli, et al.
Circulation. Cardiovascular Genetics
|
April 17, 2012
A common variant in the PTPN11 gene contributes to the risk of tetralogy of Fallot
Judith A Goodship, Darroch Hall, Ana Topf, et al.
Nature Genetics
|
March 1, 2005
Mutation in myosin heavy chain 6 causes atrial septal defect
Yung-Hao Ching, Tushar K Ghosh, Steve J Cross, et al.
Plos One
|
March 25, 2009
Genetic variation in VEGF does not contribute significantly to the risk of congenital cardiovascular malformation
Helen R Griffin, Darroch H Hall, Ana Topf, et al.
NPJ Genomic Medicine
|
June 2, 2026
Assessing the contribution of rare variants to congenital heart disease through a large-scale case-control exome study
Enrique Audain, Anna Wilsdon, Gregor Dombrowsky, et al.
Science Translational Medicine
|
May 1, 2020
CDK12 inhibition reduces abnormalities in cells from patients with myotonic dystrophy and in a mouse model
Ami Ketley, Marzena Wojciechowska, Sonja Ghidelli-Disse, et al.
Page
of 6
Search research articles
Search
Showing results (31-40 of 55) with videos related to
Sort By:
Page
of 6
Journal of Molecular and Cellular Cardiology
|
April 1, 2017
Tropomyosin 1: Multiple roles in the developing heart and in the formation of congenital heart defects
Jennifer England, Javier Granados-Riveron, Luis Polo-Parada, et al.
BMC Genetics
|
June 21, 2013
Low-frequency intermediate penetrance variants in the ROCK1 gene predispose to Tetralogy of Fallot
Julian Palomino Doza, Ana Topf, Jamie Bentham, et al.
Human Molecular Genetics
|
December 19, 2018
Transcriptome alterations in myotonic dystrophy skeletal muscle and heart
Eric T Wang, Daniel Treacy, Katy Eichinger, et al.
Human Molecular Genetics
|
November 2, 2013
High-content screening identifies small molecules that remove nuclear foci, affect MBNL distribution and CELF1 protein levels via a PKC-independent pathway in myotonic dystrophy cell lines
Ami Ketley, Catherine Z Chen, Xin Li, et al.
Genome Medicine
|
August 30, 2020
Systems genetics analysis identifies calcium-signaling defects as novel cause of congenital heart disease
Jose M G Izarzugaza, Sabrina G Ellesøe, Canan Doganli, et al.
Circulation. Cardiovascular Genetics
|
April 17, 2012
A common variant in the PTPN11 gene contributes to the risk of tetralogy of Fallot
Judith A Goodship, Darroch Hall, Ana Topf, et al.
Nature Genetics
|
March 1, 2005
Mutation in myosin heavy chain 6 causes atrial septal defect
Yung-Hao Ching, Tushar K Ghosh, Steve J Cross, et al.
Plos One
|
March 25, 2009
Genetic variation in VEGF does not contribute significantly to the risk of congenital cardiovascular malformation
Helen R Griffin, Darroch H Hall, Ana Topf, et al.
NPJ Genomic Medicine
|
June 2, 2026
Assessing the contribution of rare variants to congenital heart disease through a large-scale case-control exome study
Enrique Audain, Anna Wilsdon, Gregor Dombrowsky, et al.
Science Translational Medicine
|
May 1, 2020
CDK12 inhibition reduces abnormalities in cells from patients with myotonic dystrophy and in a mouse model
Ami Ketley, Marzena Wojciechowska, Sonja Ghidelli-Disse, et al.
Page
of 6