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Chemmedchem|March 29, 2014
Simplified silvestrol analogues with potent cytotoxic activityBill C Hawkins, Lisa M Lindqvist, Duong Nhu, et al.Cell|February 5, 2013
Bax crystal structures reveal how BH3 domains activate Bax and nucleate its oligomerization to induce apoptosisPeter E Czabotar, Dana Westphal, Grant Dewson, et al.Plos Pathogens|September 28, 2018
Infection with flaviviruses requires BCLXL for cell survivalTatsuya Suzuki, Toru Okamoto, Hiroshi Katoh, et al.Cancer Cell|November 14, 2006
The BH3 mimetic ABT-737 targets selective Bcl-2 proteins and efficiently induces apoptosis via Bak/Bax if Mcl-1 is neutralizedMark F van Delft, Andrew H Wei, Kylie D Mason, et al.Blood Advances|September 3, 2021
Outcomes of patients with CLL sequentially resistant to both BCL2 and BTK inhibitionThomas E Lew, Victor S Lin, Edward R Cliff, et al.Nature Communications|June 5, 2019
Structures of BCL-2 in complex with venetoclax reveal the molecular basis of resistance mutationsRichard W Birkinshaw, Jia-Nan Gong, Cindy S Luo, et al.Science (New York, N.Y.)|February 10, 2007
Apoptosis initiated when BH3 ligands engage multiple Bcl-2 homologs, not Bax or BakSimon N Willis, Jamie I Fletcher, Thomas Kaufmann, et al.Cell Death & Disease|August 26, 2016
MCL-1 is required throughout B-cell development and its loss sensitizes specific B-cell subsets to inhibition of BCL-2 or BCL-XLIngela B Vikström, Anne Slomp, Emma M Carrington, et al.The Journal of Biological Chemistry|September 28, 2018
CARM1-mediated methylation of protein arginine methyltransferase 5 represses human γ-globin gene expression in erythroleukemia cellsMin Nie, Yadong Wang, Chan Guo, et al.Cell Death & Disease|September 11, 2020
Defining the susceptibility of colorectal cancers to BH3-mimetic compoundsMing-Jie Luo, Michelle Palmieri, Chris D Riffkin, et al.Pageof 15