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Free Radical Biology & Medicine|November 22, 2011
Simultaneous inhibition of glutathione- and thioredoxin-dependent metabolism is necessary to potentiate 17AAG-induced cancer cell killing via oxidative stressPeter M Scarbrough, Kranti A Mapuskar, David M Mattson, et al.
Drug Resistance Updates : Reviews and Commentaries in Antimicrobial and Anticancer Chemotherapy|October 19, 2005
Redox-sensitive signaling factors as a novel molecular targets for cancer therapyJ Daniel Pennington, Tony Jau Cheng Wang, Phuongmai Nguyen, et al.
Aging|March 10, 2011
Acetylation of MnSOD directs enzymatic activity responding to cellular nutrient status or oxidative stressOzkan Ozden, Seong-Hoon Park, Hyun-Seok Kim, et al.
Free Radical Biology & Medicine|September 6, 2017
Loss of Nrf2 promotes alveolar type 2 cell loss in irradiated, fibrotic lungGeri Traver, Stacey Mont, David Gius, et al.
Free Radical Biology & Medicine|June 27, 2012
Exploring the electrostatic repulsion model in the role of Sirt3 in directing MnSOD acetylation status and enzymatic activityYueming Zhu, Seong-Hoon Park, Ozkan Ozden, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|November 17, 2007
BAG-4/SODD and associated antiapoptotic proteins are linked to aggressiveness of epithelial ovarian cancerChristina M Annunziata, Lilach Kleinberg, Ben Davidson, et al.
Molecular and Cellular Biology|September 4, 2008
BAT3 and SET1A form a complex with CTCFL/BORIS to modulate H3K4 histone dimethylation and gene expressionPhuongmai Nguyen, Gil Bar-Sela, Lunching Sun, et al.
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