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DNA Repair|November 17, 2023
TFIIH central activity in nucleotide excision repair to prevent diseaseArjan F Theil, David Häckes, Hannes LansOncogene|December 15, 2021
p53-mediated AKT and mTOR inhibition requires RFX7 and DDIT4 and depends on nutrient abundanceLuis Coronel, David Häckes, Katjana Schwab, et al.The Journal of Investigative Dermatology|June 29, 2026
Residual CSB activity explains mild UV-sensitive syndrome phenotype caused by CSB mutationsCamila Gonzalo-Hansen, David Häckes, Georgia Avramidou, et al.Nucleic Acids Research|July 1, 2021
Transcription factor RFX7 governs a tumor suppressor network in response to p53 and stressLuis Coronel, Konstantin Riege, Katjana Schwab, et al.Cell Death Discovery|March 2, 2023
Multi-omics analysis identifies RFX7 targets involved in tumor suppression and neuronal processesKatjana Schwab, Luis Coronel, Konstantin Riege, et al.NAR Cancer|December 7, 2023
Detection of oxaliplatin- and cisplatin-DNA lesions requires different global genome repair mechanisms that affect their clinical efficacyJana Slyskova, Alba Muniesa-Vargas, Israel Tojal da Silva, et al.Nature Communications|April 25, 2024
Persistent TFIIH binding to non-excised DNA damage causes cell and developmental failureAlba Muniesa-Vargas, Carlota Davó-Martínez, Cristina Ribeiro-Silva, et al.Nature Cell Biology|April 10, 2024
Transcription-coupled DNA-protein crosslink repair by CSB and CRL4CSA-mediated degradationMarjolein van Sluis, Qing Yu, Melanie van der Woude, et al.Pageof 1