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David H F Vandael

Showing results (1-10 of 9) with videos related to

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Pflugers Archiv : European Journal of Physiology|March 6, 2014
T-type channel-mediated neurotransmitter releaseEmilio Carbone, Chiara Calorio, David H F Vandael
Neuron|March 20, 2015
Excitement about inhibitory presynaptic terminalsDavid H F Vandael, Claudia Espinoza, Peter Jonas
Current Molecular Pharmacology|May 14, 2015
Cav1.3 Channels as Key Regulators of Neuron-Like Firings and Catecholamine Release in Chromaffin CellsDavid H F Vandael, Andrea Marcantoni, Emilio Carbone
The Journal of Physiology|December 28, 2016
Low pH<sub>o</sub> boosts burst firing and catecholamine release by blocking TASK-1 and BK channels while preserving Cav1 channels in mouse chromaffin cellsLaura Guarina, David H F Vandael, Valentina Carabelli, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 16, 2012
Ca(V)1.3-driven SK channel activation regulates pacemaking and spike frequency adaptation in mouse chromaffin cellsDavid H F Vandael, Annalisa Zuccotti, Joerg Striessnig, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 15, 2010
Loss of Cav1.3 channels reveals the critical role of L-type and BK channel coupling in pacemaking mouse adrenal chromaffin cellsAndrea Marcantoni, David H F Vandael, Satyajit Mahapatra, et al.
The Journal of Physiology|January 27, 2015
Reduced availability of voltage-gated sodium channels by depolarization or blockade by tetrodotoxin boosts burst firing and catecholamine release in mouse chromaffin cellsDavid H F Vandael, Matteo M Ottaviani, Christian Legros, et al.
Nature Communications|June 20, 2014
Pyrimidine-2,4,6-triones are a new class of voltage-gated L-type Ca2+ channel activatorsNadine J Ortner, Gabriella Bock, David H F Vandael, et al.
Nanotoxicology|February 17, 2011
Altered excitability of cultured chromaffin cells following exposure to multi-walled carbon nanotubesDaniela Gavello, David H F Vandael, Roberta Cesa, et al.
Pageof 1

Showing results (1-10 of 9) with videos related to

Sort By:
Pageof 1
Pflugers Archiv : European Journal of Physiology|March 6, 2014
T-type channel-mediated neurotransmitter releaseEmilio Carbone, Chiara Calorio, David H F Vandael
Neuron|March 20, 2015
Excitement about inhibitory presynaptic terminalsDavid H F Vandael, Claudia Espinoza, Peter Jonas
Current Molecular Pharmacology|May 14, 2015
Cav1.3 Channels as Key Regulators of Neuron-Like Firings and Catecholamine Release in Chromaffin CellsDavid H F Vandael, Andrea Marcantoni, Emilio Carbone
The Journal of Physiology|December 28, 2016
Low pH<sub>o</sub> boosts burst firing and catecholamine release by blocking TASK-1 and BK channels while preserving Cav1 channels in mouse chromaffin cellsLaura Guarina, David H F Vandael, Valentina Carabelli, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 16, 2012
Ca(V)1.3-driven SK channel activation regulates pacemaking and spike frequency adaptation in mouse chromaffin cellsDavid H F Vandael, Annalisa Zuccotti, Joerg Striessnig, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 15, 2010
Loss of Cav1.3 channels reveals the critical role of L-type and BK channel coupling in pacemaking mouse adrenal chromaffin cellsAndrea Marcantoni, David H F Vandael, Satyajit Mahapatra, et al.
The Journal of Physiology|January 27, 2015
Reduced availability of voltage-gated sodium channels by depolarization or blockade by tetrodotoxin boosts burst firing and catecholamine release in mouse chromaffin cellsDavid H F Vandael, Matteo M Ottaviani, Christian Legros, et al.
Nature Communications|June 20, 2014
Pyrimidine-2,4,6-triones are a new class of voltage-gated L-type Ca2+ channel activatorsNadine J Ortner, Gabriella Bock, David H F Vandael, et al.
Nanotoxicology|February 17, 2011
Altered excitability of cultured chromaffin cells following exposure to multi-walled carbon nanotubesDaniela Gavello, David H F Vandael, Roberta Cesa, et al.
Pageof 1