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Sub-Cellular Biochemistry|December 11, 2012
Mechanisms of transthyretin aggregation and toxicityRobert J Gasperini, David W Klaver, Xu Hou, et al.
Neuro-Degenerative Diseases|March 13, 2010
Effect of heparin on APP metabolism and Abeta production in cortical neuronsDavid Klaver, Amos C Hung, Robert Gasperini, et al.
Neurobiology of Aging|July 18, 2008
Memory loss caused by beta-amyloid protein is rescued by a beta(3)-adrenoceptor agonistMarie E Gibbs, Danuta Maksel, Zoe Gibbs, et al.
Journal of Neurochemistry|August 23, 2012
Size and sulfation are critical for the effect of heparin on APP processing and Aβ productionHao Cui, Amos C Hung, Craig Freeman, et al.
Brain Research|February 20, 2003
Acetylcholinesterase is increased in mouse neuronal and astrocyte cultures after treatment with beta-amyloid peptidesJavier Sáez-Valero, Lisa R Fodero, Anthony R White, et al.
IUBMB Life|July 3, 2004
Alzheimer's disease therapeutics: new approaches to an ageing problemDavid H Small, Dusan Losic, Lisandra L Martin, et al.
European Journal of Biochemistry|June 20, 2002
Toxicity of substrate-bound amyloid peptides on vascular smooth muscle cells is enhanced by homocysteineSu San Mok, Bradley J Turner, Konrad Beyreuther, et al.
Journal of Neurochemistry|November 2, 2006
Transthyretin oligomers induce calcium influx via voltage-gated calcium channelsXu Hou, Helena C Parkington, Harold A Coleman, et al.
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