Showing results (61-70 of 115) with videos related to

Sort By:
Pageof 12
The Journal of Clinical Investigation|September 20, 2008
GSK-3beta in mouse fibroblasts controls wound healing and fibrosis through an endothelin-1-dependent mechanismMohit Kapoor, Shangxi Liu, Xu Shi-wen, et al.
The Journal of Investigative Dermatology|May 7, 2010
Epithelial cells promote fibroblast activation via IL-1alpha in systemic sclerosisNima Aden, Anna Nuttall, Xu Shiwen, et al.
Cells|July 14, 2023
Single-Cell Analysis of ADSC Interactions with Fibroblasts and Endothelial Cells in Scleroderma SkinMarvin L Frommer, Benjamin J Langridge, Laura Awad, et al.
The Journal of Investigative Dermatology|June 10, 2011
Loss of PTEN expression by dermal fibroblasts causes skin fibrosisSunil K Parapuram, Xu Shi-wen, Christopher Elliott, et al.
American Journal of Respiratory and Critical Care Medicine|August 17, 2010
An essential role for resident fibroblasts in experimental lung fibrosis is defined by lineage-specific deletion of high-affinity type II transforming growth factor β receptorRachel K Hoyles, Emma C Derrett-Smith, Korsa Khan, et al.
Arthritis and Rheumatism|January 1, 2009
Requirement of transforming growth factor beta-activated kinase 1 for transforming growth factor beta-induced alpha-smooth muscle actin expression and extracellular matrix contraction in fibroblastsXu Shi-wen, Sunil K Parapuram, Daphne Pala, et al.
Fibrogenesis & Tissue Repair|April 2, 2011
Thrombospondin 1 is a key mediator of transforming growth factor β-mediated cell contractility in systemic sclerosis via a mitogen-activated protein kinase kinase (MEK)/extracellular signal-regulated kinase (ERK)-dependent mechanismYunliang Chen, Andrew Leask, David J Abraham, et al.
Fibrogenesis & Tissue Repair|March 17, 2015
Erratum to: Thrombospondin 1 is a key mediator of transforming growth factor b-mediated cell contractility in systemic sclerosis via a mitogen-activated protein kinase kinase (MEK)/extracellular signal-regulated kinase (ERK)-dependent mechanismYunliang Chen, Andrew Leask, David J Abraham, et al.
Molecular Biology of the Cell|April 6, 2007
FAK is required for TGFbeta-induced JNK phosphorylation in fibroblasts: implications for acquisition of a matrix-remodeling phenotypeShangxi Liu, Shi-wen Xu, Laura Kennedy, et al.
Plos One|July 18, 2019
Stem cell enriched lipotransfer reverses the effects of fibrosis in systemic sclerosisAurora Almadori, Michelle Griffin, Caroline M Ryan, et al.
Pageof 12