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The American Journal of Pathology|October 7, 2004
ApoAI deficiency results in marked reductions in plasma cholesterol but no alterations in amyloid-beta pathology in a mouse model of Alzheimer's disease-like cerebral amyloidosisAnne M Fagan, Erin Christopher, Jennie W Taylor, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 28, 2014
Proteopathic tau seeding predicts tauopathy in vivoBrandon B Holmes, Jennifer L Furman, Thomas E Mahan, et al.
JAMA Neurology|August 21, 2013
Cerebrospinal fluid Aβ42, phosphorylated Tau181, and resting-state functional connectivityLiang Wang, Matthew R Brier, Abraham Z Snyder, et al.
ACS Chemical Neuroscience|February 24, 2017
Disease-Modifying Effects of M1 Muscarinic Acetylcholine Receptor Activation in an Alzheimer's Disease Mouse ModelEvan P Lebois, Jason P Schroeder, Thomas J Esparza, et al.
Scientific Reports|August 27, 2017
Diversity of Amyloid-beta Proteoforms in the Alzheimer's Disease BrainNorelle C Wildburger, Thomas J Esparza, Richard D LeDuc, et al.
Neuroimage|August 24, 2013
Disrupted modular organization of resting-state cortical functional connectivity in U.S. military personnel following concussive 'mild' blast-related traumatic brain injuryKihwan Han, Christine L Mac Donald, Ann M Johnson, et al.
Molecular Neurodegeneration|April 27, 2012
Multiple γ-secretase product peptides are coordinately increased in concentration in the cerebrospinal fluid of a subpopulation of sporadic Alzheimer's disease subjectsSaori Hata, Miyako Taniguchi, Yi Piao, et al.
The Lancet. Neurology|September 10, 2013
Preclinical Alzheimer's disease and its outcome: a longitudinal cohort studyStephanie Jb Vos, Chengjie Xiong, Pieter Jelle Visser, et al.
Annals of Clinical and Translational Neurology|August 23, 2016
Nmnat1 protects neuronal function without altering phospho-tau pathology in a mouse model of tauopathyErik S Musiek, David D Xiong, Tirth Patel, et al.
Analytical Biochemistry|April 18, 2023
Apolipoprotein E O-glycosylation is associated with amyloid plaques and APOE genotypePaige E Lawler, James G Bollinger, Suzanne E Schindler, et al.
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