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Cancer|April 30, 2009
Circulating biomarkers for vascular endothelial growth factor inhibitors in renal cell carcinomaAmado J Zurita, Eric Jonasch, Hua-Kang Wu, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|August 15, 2022
Molecular Mechanisms and Future Implications of VEGF/VEGFR in Cancer TherapySonia A Patel, Monique B Nilsson, Xiuning Le, et al.
Lung Cancer (Amsterdam, Netherlands)|February 25, 2021
Controversies and challenges in the pathologic examination of lung resection specimens after neoadjuvant treatmentAnnikka Weissferdt, Apar Pataer, Stephen G Swisher, et al.
Cancer Research|September 27, 2007
Meeting Report: Fifth Annual AACR Frontiers in Cancer Prevention ResearchAshraful Hoque, Howard L Parnes, Michael E Stefanek, et al.
Plos One|August 28, 2015
8-Chloroadenosine Sensitivity in Renal Cell Carcinoma Is Associated with AMPK Activation and mTOR Pathway InhibitionAlper Y Kearney, You-Hong Fan, Uma Giri, et al.
Clinical Lung Cancer|November 25, 2021
Design and Rationale for a Phase III, Double-Blind, Placebo-Controlled Study of Neoadjuvant Durvalumab + Chemotherapy Followed by Adjuvant Durvalumab for the Treatment of Patients With Resectable Stages II and III non-small-cell Lung Cancer: The AEGEAN TrialJohn V Heymach, Tetsuya Mitsudomi, David Harpole, et al.
Cell Cycle (Georgetown, Tex.)|November 26, 2008
Specific blockade of VEGF and HER2 pathways results in greater growth inhibition of breast cancer xenografts that overexpress HER2Xiao-Feng Le, Weiqun Mao, Chunhua Lu, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|November 24, 2006
Vascular endothelial growth factor accelerates compensatory lung growth after unilateral pneumonectomyMaromi K Sakurai, Sang Lee, Danielle A Arsenault, et al.
Scientific Reports|May 28, 2016
Different dietary patterns and reduction of lung cancer risk: A large case-control study in the U.SHuakang Tu, John V Heymach, Chi-Pang Wen, et al.
Neuro-Oncology|September 12, 2012
Glioblastoma resistance to anti-VEGF therapy is associated with myeloid cell infiltration, stem cell accumulation, and a mesenchymal phenotypeYuji Piao, Ji Liang, Lindsay Holmes, et al.
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