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Journal of Cell Science|March 18, 2003
Extracellular Ca(2+) sensing contributes to excess Ca(2+) accumulation and vacuolar fragmentation in a pmr1Delta mutant of S. cerevisiaeRichard Kellermayer, David P Aiello, Attila Miseta, et al.
RNA (New York, N.Y.)|March 31, 2009
Connection between stop codon reassignment and frequent use of shifty stop frameshiftingHaritha Vallabhaneni, Hua Fan-Minogue, David M Bedwell, et al.
Eukaryotic Cell|June 11, 2003
A Saccharomyces cerevisiae mutant unable to convert glucose to glucose-6-phosphate accumulates excessive glucose in the endoplasmic reticulum due to core oligosaccharide trimmingAttila Miseta, Margit Tökés-Füzesi, David P Aiello, et al.
Critical Reviews in Biochemistry and Molecular Biology|June 8, 2012
Suppression of premature termination codons as a therapeutic approachKim M Keeling, Dan Wang, Sara E Conard, et al.
The Journal of Biological Chemistry|July 15, 2004
The Ca2+ homeostasis defects in a pgm2Delta strain of Saccharomyces cerevisiae are caused by excessive vacuolar Ca2+ uptake mediated by the Ca2+-ATPase Pmc1pDavid P Aiello, Lianwu Fu, Attila Miseta, et al.
Molecular and Cellular Biology|July 1, 2006
Tpa1p is part of an mRNP complex that influences translation termination, mRNA deadenylation, and mRNA turnover in Saccharomyces cerevisiaeKim M Keeling, Joe Salas-Marco, Lev Z Osherovich, et al.
The Journal of Biological Chemistry|January 13, 2009
Poly-L-aspartic acid enhances and prolongs gentamicin-mediated suppression of the CFTR-G542X mutation in a cystic fibrosis mouse modelMing Du, Kim M Keeling, Liming Fan, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 15, 2008
PTC124 is an orally bioavailable compound that promotes suppression of the human CFTR-G542X nonsense allele in a CF mouse modelMing Du, Xiaoli Liu, Ellen M Welch, et al.
Molecular Microbiology|May 25, 2002
Hexose phosphorylation and the putative calcium channel component Mid1p are required for the hexose-induced transient elevation of cytosolic calcium response in Saccharomyces cerevisiaeMargit Tökés-Füzesi, David M Bedwell, Imre Repa, et al.
Journal of Molecular Medicine (Berlin, Germany)|March 17, 2006
Clinical doses of amikacin provide more effective suppression of the human CFTR-G542X stop mutation than gentamicin in a transgenic CF mouse modelMing Du, Kim M Keeling, Liming Fan, et al.
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