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Cell Reports|June 17, 2014
A conditional system to specifically link disruption of protein-coding function with reporter expression in miceShin-Heng Chiou, Caroline Kim-Kiselak, Viviana I Risca, et al.Nature Communications|March 1, 2022
LKB1 drives stasis and C/EBP-mediated reprogramming to an alveolar type II fate in lung cancerChristopher W Murray, Jennifer J Brady, Mingqi Han, et al.Communications Biology|March 10, 2023
Setd2 inactivation sensitizes lung adenocarcinoma to inhibitors of oxidative respiration and mTORC1 signalingDavid M Walter, Amy C Gladstein, Katherine R Doerig, et al.Nature|May 3, 2019
RB constrains lineage fidelity and multiple stages of tumour progression and metastasisDavid M Walter, Travis J Yates, Miguel Ruiz-Torres, et al.Nature Cell Biology|August 3, 2021
LKB1 inactivation modulates chromatin accessibility to drive metastatic progressionSarah E Pierce, Jeffrey M Granja, M Ryan Corces, et al.Plos Genetics|December 8, 2021
RB depletion is required for the continuous growth of tumors initiated by loss of RBAlex Doan, Julia Arand, Diana Gong, et al.Nature Aging|November 4, 2025
Aging represses oncogenic KRAS-driven lung tumorigenesis and alters tumor suppressionEmily G Shuldiner, Saswati Karmakar, Min K Tsai, et al.Biorxiv : the Preprint Server for Biology|June 12, 2025
The H3 K36M oncohistone inhibits NSD2 to activate a SETD2-dependent antiviral-like immune response in KRAS-driven lung cancerAmy C Gladstein, Carson D Poltorack, Akino Mercy Charles Solomon, et al.Nature|April 8, 2011
Suppression of lung adenocarcinoma progression by Nkx2-1Monte M Winslow, Talya L Dayton, Roel G W Verhaak, et al.Nature Communications|July 21, 2023
p53 restoration in small cell lung cancer identifies a latent cyclophilin-dependent necrosis mechanismJonuelle Acosta, Qinglan Li, Nelson F Freeburg, et al.Pageof 3