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Proceedings of the National Academy of Sciences of the United States of America|February 16, 2005
Control of antiviral defenses through hepatitis C virus disruption of retinoic acid-inducible gene-I signalingEileen Foy, Kui Li, Rhea Sumpter, et al.Cell|April 4, 2017
RIPK3 Restricts Viral Pathogenesis via Cell Death-Independent NeuroinflammationBrian P Daniels, Annelise G Snyder, Tayla M Olsen, et al.Journal of Immunology (Baltimore, Md. : 1950)|August 24, 2005
Shared and unique functions of the DExD/H-box helicases RIG-I, MDA5, and LGP2 in antiviral innate immunityMitsutoshi Yoneyama, Mika Kikuchi, Kanae Matsumoto, et al.Journal of Virology|October 19, 2007
Distinct RIG-I and MDA5 signaling by RNA viruses in innate immunityYueh-Ming Loo, Jamie Fornek, Nanette Crochet, et al.The Journal of Infectious Diseases|December 28, 2020
SARS-CoV-2 Serologic Assays in Control and Unknown Populations Demonstrate the Necessity of Virus Neutralization TestingJennifer A Rathe, Emily A Hemann, Julie Eggenberger, et al.Journal of Immunology (Baltimore, Md. : 1950)|May 8, 2020
Systematic Profiling of Full-Length Ig and TCR Repertoire Diversity in Rhesus Macaque through Long Read Transcriptome SequencingHayden N Brochu, Elizabeth Tseng, Elise Smith, et al.Plos One|October 18, 2017
Rapid reversal of innate immune dysregulation in blood of patients and livers of humanized mice with HCV following DAA therapyMatthew A Burchill, Justin A Roby, Nanette Crochet, et al.Plos Pathogens|April 19, 2014
Deficient IFN signaling by myeloid cells leads to MAVS-dependent virus-induced sepsisAmelia K Pinto, Hilario J Ramos, Xiaobo Wu, et al.The Journal of Clinical Investigation|January 31, 2017
Regional astrocyte IFN signaling restricts pathogenesis during neurotropic viral infectionBrian P Daniels, Harsha Jujjavarapu, Douglas M Durrant, et al.Journal of Virology|January 13, 2017
MAVS Is Essential for Primary CD4+ T Cell Immunity but Not for Recall T Cell Responses following an Attenuated West Nile Virus InfectionHuanle Luo, Evandro Winkelmann, Guorui Xie, et al.Pageof 34