Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

David M Thomson

Showing results (21-30 of 43) with videos related to

Pageof 5
Sort By:
Scientific Reports|July 25, 2020
Drug-responsive autism phenotypes in the 16p11.2 deletion mouse model: a central role for gene-environment interactionsEmma J Mitchell, David M Thomson, Rebecca L Openshaw, et al.
Communications Biology|May 24, 2023
16p11.2 deletion mice exhibit compromised fronto-temporal connectivity, GABAergic dysfunction, and enhanced attentional abilityRebecca L Openshaw, David M Thomson, Greg C Bristow, et al.
Cell Reports|April 23, 2020
16p11 Duplication Disrupts Hippocampal-Orbitofrontal-Amygdala Connectivity, Revealing a Neural Circuit Endophenotype for SchizophreniaGreg C Bristow, David M Thomson, Rebecca L Openshaw, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|September 10, 2011
Characterization of the liver kinase B1-mouse protein-25 -Ste-20-related adaptor protein complex in adult mouse skeletal muscleCody D Smith, Richard A Compton, Joshua S Bowler, et al.
American Journal of Physiology. Endocrinology and Metabolism|March 20, 2019
Multitissue analysis of exercise and metformin on doxorubicin-induced iron dysregulationAmy D Mackay, Erik D Marchant, Devin J Munk, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|June 25, 2011
Reductions in RIP140 are not required for exercise- and AICAR-mediated increases in skeletal muscle mitochondrial contentBruce C Frier, Chad R Hancock, Jonathan P Little, et al.
Biochemical Society Transactions|April 8, 2015
The zDHHC family of S-acyltransferasesKimon Lemonidis, Martin W Werno, Jennifer Greaves, et al.
Journal of Lipid Research|February 26, 2014
BRCA1 is a novel regulator of metabolic function in skeletal muscleKathryn C Jackson, Eva-Karin Gidlund, Jessica Norrbom, et al.
FASEB Bioadvances|March 10, 2025
Chronic treatment of old mice with AICAR reverses age-related changes in exercise performance and skeletal muscle gene expressionShalene H Wilcox, Jouber Calixto, Steven D Dray, et al.
Genes, Brain, and Behavior|October 20, 2020
Impaired working memory, cognitive flexibility and reward processing in mice genetically lacking Gpr88: Evidence for a key role for Gpr88 in multiple cortico-striatal-thalamic circuitsDavid M Thomson, Rebecca L Openshaw, Emma J Mitchell, et al.
Pageof 5

Showing results (21-30 of 43) with videos related to

Sort By:
Pageof 5
Scientific Reports|July 25, 2020
Drug-responsive autism phenotypes in the 16p11.2 deletion mouse model: a central role for gene-environment interactionsEmma J Mitchell, David M Thomson, Rebecca L Openshaw, et al.
Communications Biology|May 24, 2023
16p11.2 deletion mice exhibit compromised fronto-temporal connectivity, GABAergic dysfunction, and enhanced attentional abilityRebecca L Openshaw, David M Thomson, Greg C Bristow, et al.
Cell Reports|April 23, 2020
16p11 Duplication Disrupts Hippocampal-Orbitofrontal-Amygdala Connectivity, Revealing a Neural Circuit Endophenotype for SchizophreniaGreg C Bristow, David M Thomson, Rebecca L Openshaw, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|September 10, 2011
Characterization of the liver kinase B1-mouse protein-25 -Ste-20-related adaptor protein complex in adult mouse skeletal muscleCody D Smith, Richard A Compton, Joshua S Bowler, et al.
American Journal of Physiology. Endocrinology and Metabolism|March 20, 2019
Multitissue analysis of exercise and metformin on doxorubicin-induced iron dysregulationAmy D Mackay, Erik D Marchant, Devin J Munk, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|June 25, 2011
Reductions in RIP140 are not required for exercise- and AICAR-mediated increases in skeletal muscle mitochondrial contentBruce C Frier, Chad R Hancock, Jonathan P Little, et al.
Biochemical Society Transactions|April 8, 2015
The zDHHC family of S-acyltransferasesKimon Lemonidis, Martin W Werno, Jennifer Greaves, et al.
Journal of Lipid Research|February 26, 2014
BRCA1 is a novel regulator of metabolic function in skeletal muscleKathryn C Jackson, Eva-Karin Gidlund, Jessica Norrbom, et al.
FASEB Bioadvances|March 10, 2025
Chronic treatment of old mice with AICAR reverses age-related changes in exercise performance and skeletal muscle gene expressionShalene H Wilcox, Jouber Calixto, Steven D Dray, et al.
Genes, Brain, and Behavior|October 20, 2020
Impaired working memory, cognitive flexibility and reward processing in mice genetically lacking Gpr88: Evidence for a key role for Gpr88 in multiple cortico-striatal-thalamic circuitsDavid M Thomson, Rebecca L Openshaw, Emma J Mitchell, et al.
Pageof 5