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Frontiers in Bioscience : a Journal and Virtual Library|April 18, 2003
Repair mechanisms for oxidative DNA damageDavid M Wilson, Troy M Sofinowski, Daniel R McNeillMolecular Cancer Research : MCR|January 30, 2007
A dominant-negative form of the major human abasic endonuclease enhances cellular sensitivity to laboratory and clinical DNA-damaging agentsDaniel R McNeill, David M WilsonJournal of Molecular Biology|April 29, 2008
Characterization of abasic endonuclease activity of human Ape1 on alternative substrates, as well as effects of ATP and sequence context on AP site incisionBrian R Berquist, Daniel R McNeill, David M WilsonEnvironmental Health Perspectives|May 26, 2004
Inhibition of Ape1 nuclease activity by lead, iron, and cadmiumDaniel R McNeill, Avinash Narayana, Heng-Kuan Wong, et al.Molecular Carcinogenesis|October 3, 2006
Lead promotes abasic site accumulation and co-mutagenesis in mammalian cells by inhibiting the major abasic endonuclease Ape1Daniel R McNeill, Heng-Kuan Wong, Avinash Narayana, et al.The Journal of Biological Chemistry|July 3, 2015
Reduced Nuclease Activity of Apurinic/Apyrimidinic Endonuclease (APE1) Variants on Nucleosomes: IDENTIFICATION OF ACCESS RESIDUESJohn M Hinz, Peng Mao, Daniel R McNeill, et al.Nucleic Acids Research|August 7, 2008
XRCC1 protects against the lethality of induced oxidative DNA damage in nondividing neural cellsAvanti Kulkarni, Daniel R McNeill, Marc Gleichmann, et al.Biochemistry|October 26, 2005
DNA damage levels and biochemical repair capacities associated with XRCC1 deficiencyHeng-Kuan Wong, Daemyung Kim, Barbara A Hogue, et al.Molecular Cancer Research : MCR|May 28, 2009
Impairment of APE1 function enhances cellular sensitivity to clinically relevant alkylators and antimetabolitesDaniel R McNeill, Wing Lam, Theodore L DeWeese, et al.Scientific Reports|October 13, 2017
Development of a Cell-Based Assay for Measuring Base Excision Repair ResponsesTyler Golato, Boris Brenerman, Daniel R McNeill, et al.Pageof 32