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Proceedings of the National Academy of Sciences of the United States of America|July 13, 2004
Ethanol toxicity in pancreatic acinar cells: mediation by nonoxidative fatty acid metabolitesDavid N Criddle, Michael G T Raraty, John P Neoptolemos, et al.
Pancreas|November 19, 2015
Translational Insights Into Peroxisome Proliferator-Activated Receptors in Experimental Acute PancreatitisWei Huang, Peter Szatmary, Meihua Wan, et al.
Gastroenterology|March 15, 2006
Fatty acid ethyl esters cause pancreatic calcium toxicity via inositol trisphosphate receptors and loss of ATP synthesisDavid N Criddle, John Murphy, Gregorio Fistetto, et al.
Life Sciences|September 18, 2007
Effects of anethole and structural analogues on the contractility of rat isolated aorta: Involvement of voltage-dependent Ca2+-channelsPedro Marcos G Soares, Ricardo F Lima, Alana de Freitas Pires, et al.
American Journal of Physiology. Gastrointestinal and Liver Physiology|October 10, 2009
Cholecystokinin-58 and cholecystokinin-8 exhibit similar actions on calcium signaling, zymogen secretion, and cell fate in murine pancreatic acinar cellsDavid N Criddle, David M Booth, Rajarshi Mukherjee, et al.
The Journal of Pharmacy and Pharmacology|November 5, 2004
Red marine alga Bryothamnion triquetrum lectin induces endothelium-dependent relaxation of the rat aorta via release of nitric oxideRicardo F Lima, David N Criddle, Emmanuel P Souza, et al.
Pancreatology : Official Journal of the International Association of Pancreatology (IAP) ... [Et Al.]|June 27, 2017
Selective inhibition of BET proteins reduces pancreatic damage and systemic inflammation in bile acid- and fatty acid ethyl ester- but not caerulein-induced acute pancreatitisWei Huang, Andrea C Haynes, Rajarshi Mukherjee, et al.
The Journal of Physiology|May 3, 2018
Intracellular rupture, exocytosis and actin interaction of endocytic vacuoles in pancreatic acinar cells: initiating events in acute pancreatitisMichael Chvanov, Francesca De Faveri, Danielle Moore, et al.
Frontiers in Physiology|July 28, 2018
F1F0-ATP Synthase Inhibitory Factor 1 in the Normal Pancreas and in Pancreatic Ductal Adenocarcinoma: Effects on Bioenergetics, Invasion and ProliferationHelen Tanton, Svetlana Voronina, Anthony Evans, et al.
The Journal of Biological Chemistry|April 8, 2018
Oxidative stress alters mitochondrial bioenergetics and modifies pancreatic cell death independently of cyclophilin D, resulting in an apoptosis-to-necrosis shiftJane A Armstrong, Nicole J Cash, Yulin Ouyang, et al.
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