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David Neary

Showing results (51-60 of 73) with videos related to

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Acta Neuropathologica|January 19, 2011
Granular expression of prolyl-peptidyl isomerase PIN1 is a constant and specific feature of Alzheimer's disease pathology and is independent of tau, Aβ and TDP-43 pathologyAyoub Dakson, Osamu Yokota, Margaret Esiri, et al.
Alzheimer Disease and Associated Disorders|October 8, 2014
Semantic Corticobasal Dementia: Challenging Nosology in Frontotemporal Lobe DegenerationSimona Luzzi, Katia Fabi, Viviana Cafazzo, et al.
Cortex; a Journal Devoted to the Study of the Nervous System and Behavior|October 19, 2007
Cognitive phenotypes in Alzheimer's disease and genetic riskJulie S Snowden, Cheryl L Stopford, Camille L Julien, et al.
Neuroscience Letters|January 25, 2006
CHMP2B mutations are not a common cause of frontotemporal lobar degenerationAshley Cannon, Matthew Baker, Brad Boeve, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|April 4, 2015
Psychosis associated with expansions in the C9orf72 gene: the influence of a 10 base pair gene deletionJulie S Snowden, Jennifer Harris, Jennifer Adams, et al.
American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics : the Official Publication of the International Society of Psychiatric Genetics|February 3, 2004
Evidence of a founder effect in families with frontotemporal dementia that harbor the tau +16 splice mutationStuart Pickering-Brown, Matt Baker, Thomas Bird, et al.
Neurobiology of Aging|March 14, 2012
Analysis of the hexanucleotide repeat in C9ORF72 in Alzheimer's diseaseSara Rollinson, Nicola Halliwell, Kate Young, et al.
Journal of Nuclear Medicine : Official Publication, Society of Nuclear Medicine|February 7, 2015
18F-florbetapir PET in patients with frontotemporal dementia and Alzheimer diseaseChristopher Kobylecki, Tobias Langheinrich, Rainer Hinz, et al.
Acta Neuropathologica|March 23, 2011
The most common type of FTLD-FUS (aFTLD-U) is associated with a distinct clinical form of frontotemporal dementia but is not related to mutations in the FUS geneJulie S Snowden, Quan Hu, Sara Rollinson, et al.
Acta Neuropathologica|October 14, 2005
Histopathological changes underlying frontotemporal lobar degeneration with clinicopathological correlationJing Shi, Catherine L Shaw, Daniel Du Plessis, et al.
Pageof 8

Showing results (51-60 of 73) with videos related to

Sort By:
Pageof 8
Acta Neuropathologica|January 19, 2011
Granular expression of prolyl-peptidyl isomerase PIN1 is a constant and specific feature of Alzheimer's disease pathology and is independent of tau, Aβ and TDP-43 pathologyAyoub Dakson, Osamu Yokota, Margaret Esiri, et al.
Alzheimer Disease and Associated Disorders|October 8, 2014
Semantic Corticobasal Dementia: Challenging Nosology in Frontotemporal Lobe DegenerationSimona Luzzi, Katia Fabi, Viviana Cafazzo, et al.
Cortex; a Journal Devoted to the Study of the Nervous System and Behavior|October 19, 2007
Cognitive phenotypes in Alzheimer's disease and genetic riskJulie S Snowden, Cheryl L Stopford, Camille L Julien, et al.
Neuroscience Letters|January 25, 2006
CHMP2B mutations are not a common cause of frontotemporal lobar degenerationAshley Cannon, Matthew Baker, Brad Boeve, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|April 4, 2015
Psychosis associated with expansions in the C9orf72 gene: the influence of a 10 base pair gene deletionJulie S Snowden, Jennifer Harris, Jennifer Adams, et al.
American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics : the Official Publication of the International Society of Psychiatric Genetics|February 3, 2004
Evidence of a founder effect in families with frontotemporal dementia that harbor the tau +16 splice mutationStuart Pickering-Brown, Matt Baker, Thomas Bird, et al.
Neurobiology of Aging|March 14, 2012
Analysis of the hexanucleotide repeat in C9ORF72 in Alzheimer's diseaseSara Rollinson, Nicola Halliwell, Kate Young, et al.
Journal of Nuclear Medicine : Official Publication, Society of Nuclear Medicine|February 7, 2015
18F-florbetapir PET in patients with frontotemporal dementia and Alzheimer diseaseChristopher Kobylecki, Tobias Langheinrich, Rainer Hinz, et al.
Acta Neuropathologica|March 23, 2011
The most common type of FTLD-FUS (aFTLD-U) is associated with a distinct clinical form of frontotemporal dementia but is not related to mutations in the FUS geneJulie S Snowden, Quan Hu, Sara Rollinson, et al.
Acta Neuropathologica|October 14, 2005
Histopathological changes underlying frontotemporal lobar degeneration with clinicopathological correlationJing Shi, Catherine L Shaw, Daniel Du Plessis, et al.
Pageof 8