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Neural Regeneration Research|October 7, 2022
Potential physiological and pathological roles for axonal ryanodine receptorsDavid P StirlingTrends in Molecular Medicine|March 9, 2010
Mechanisms of axonal injury: internodal nanocomplexes and calcium deregulationDavid P Stirling, Peter K StysJournal of Neuroscience Research|April 29, 2008
Dynamics of the inflammatory response after murine spinal cord injury revealed by flow cytometryDavid P Stirling, V Wee YongJournal of Neurotrauma|December 16, 2021
Inhibiting Calcium Release from Ryanodine Receptors Protects Axons after Spinal Cord InjuryBen C Orem, Arezoo Rajaee, David P StirlingNeurobiology of Disease|October 4, 2020
IP<sub>3</sub>R-mediated intra-axonal Ca<sup>2+</sup> release contributes to secondary axonal degeneration following contusive spinal cord injuryBen C Orem, Arezoo Rajaee, David P StirlingNeurobiology of Disease|December 18, 2019
Inhibiting store-operated calcium entry attenuates white matter secondary degeneration following SCIBen C Orem, Steven B Partain, David P StirlingExperimental Neurology|July 15, 2014
Immune modulatory therapies for spinal cord injury--past, present and futureJason R Plemel, V Wee Yong, David P StirlingNeurotherapeutics : the Journal of the American Society for Experimental Neurotherapeutics|October 9, 2007
Targeting MMPs in acute and chronic neurological conditionsV Wee Yong, Smriti M Agrawal, David P StirlingThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 23, 2009
Depletion of Ly6G/Gr-1 leukocytes after spinal cord injury in mice alters wound healing and worsens neurological outcomeDavid P Stirling, Shuhong Liu, Paul Kubes, et al.Neural Regeneration Research|July 14, 2023
Ca<sup>2+</sup>-induced myelin pathology precedes axonal spheroid formation and is mediated in part by store-operated Ca<sup>2+</sup> entry after spinal cord injurySpencer Ames, Kia Adams, Mariah E Geisen, et al.Pageof 3