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Journal of Virology|December 3, 2010
A systematic analysis of human papillomavirus (HPV) E6 PDZ substrates identifies MAGI-1 as a major target of HPV type 16 (HPV-16) and HPV-18 whose loss accompanies disruption of tight junctionsChristian Kranjec, Lawrence BanksMutation Research. Reviews in Mutation Research|May 23, 2017
Molecular mechanisms underlying human papillomavirus E6 and E7 oncoprotein-induced cell transformationSuruchi Mittal, Lawrence BanksJournal of Virology|August 25, 2017
Human Papillomavirus 16 (HPV-16), HPV-18, and HPV-31 E6 Override the Normal Phosphoregulation of E6AP Enzymatic ActivityJayashree Thatte, Lawrence BanksJournal of Virology|November 2, 2012
SNX17 facilitates infection with diverse papillomavirus typesMartina Bergant, Lawrence BanksThe Journal of Biological Chemistry|August 7, 2003
Regulation of the discs large tumor suppressor by a phosphorylation-dependent interaction with the beta-TrCP ubiquitin ligase receptorFiamma Mantovani, Lawrence BanksViruses|October 26, 2021
The Not-So-Good, the Bad and the Ugly: HPV E5, E6 and E7 Oncoproteins in the Orchestration of CarcinogenesisOm Basukala, Lawrence BanksMethods in Molecular Medicine|December 15, 2005
Transformation assays for HPV oncoproteinsPaola Massimi, Lawrence BanksPlos Pathogens|September 21, 2016
Interaction of the Human Papillomavirus E6 Oncoprotein with Sorting Nexin 27 Modulates Endocytic Cargo Transport PathwaysKetaki Ganti, Paola Massimi, Joaquin Manzo-Merino, et al.The Journal of General Virology|February 28, 2019
Acquisition of a phospho-acceptor site enhances HPV E6 PDZ-binding motif functional promiscuityVanessa Sarabia-Vega, Lawrence BanksAntiviral Therapy|November 13, 2004
HPV proteins as targets for therapeutic interventionHelena Sterlinko Grm, Lawrence BanksPageof 18