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David R Borchelt

Showing results (21-30 of 159) with videos related to

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Biological Psychiatry|May 17, 2017
Prion-like Spreading in TauopathiesJacob I Ayers, Benoit I Giasson, David R Borchelt
Molecular Neurodegeneration|February 16, 2021
Remodeling Alzheimer-amyloidosis models by seedingBrittany S Ulm, David R Borchelt, Brenda D Moore
Journal of Neurochemistry|December 17, 2008
Modulation of mutant superoxide dismutase 1 aggregation by co-expression of wild-type enzymeMercedes Prudencio, Armando Durazo, Julian P Whitelegge, et al.
Journal of Neuropathology and Experimental Neurology|December 20, 2007
Limited clearance of pre-existing amyloid plaques after intracerebral injection of Abeta antibodies in two mouse models of Alzheimer diseaseStina M Fangmark Tucker, David R Borchelt, Juan C Troncoso
Plos One|January 7, 2014
An analysis of interactions between fluorescently-tagged mutant and wild-type SOD1 in intracellular inclusionsDavid A Qualls, Keith Crosby, Hilda Brown, et al.
Neurobiology of Aging|June 20, 2015
Behavioral abnormalities in APPSwe/PS1dE9 mouse model of AD-like pathology: comparative analysis across multiple behavioral domainsChristopher Janus, Abigail Y Flores, Guilian Xu, et al.
Human Molecular Genetics|September 28, 2010
An examination of wild-type SOD1 in modulating the toxicity and aggregation of ALS-associated mutant SOD1Mercedes Prudencio, Armando Durazo, Julian P Whitelegge, et al.
Nature Neuroscience|June 27, 2002
Genetically engineered mouse models of neurodegenerative diseasesPhilip C Wong, Huaibin Cai, David R Borchelt, et al.
Alzheimer'S Research & Therapy|April 28, 2012
Reduction of low-density lipoprotein receptor-related protein (LRP1) in hippocampal neurons does not proportionately reduce, or otherwise alter, amyloid deposition in APPswe/PS1dE9 transgenic miceGuilian Xu, Cameron C Green, Susan E Fromholt, et al.
Human Molecular Genetics|June 2, 2009
Variation in aggregation propensities among ALS-associated variants of SOD1: correlation to human diseaseMercedes Prudencio, P John Hart, David R Borchelt, et al.
Pageof 16

Showing results (21-30 of 159) with videos related to

Sort By:
Pageof 16
Biological Psychiatry|May 17, 2017
Prion-like Spreading in TauopathiesJacob I Ayers, Benoit I Giasson, David R Borchelt
Molecular Neurodegeneration|February 16, 2021
Remodeling Alzheimer-amyloidosis models by seedingBrittany S Ulm, David R Borchelt, Brenda D Moore
Journal of Neurochemistry|December 17, 2008
Modulation of mutant superoxide dismutase 1 aggregation by co-expression of wild-type enzymeMercedes Prudencio, Armando Durazo, Julian P Whitelegge, et al.
Journal of Neuropathology and Experimental Neurology|December 20, 2007
Limited clearance of pre-existing amyloid plaques after intracerebral injection of Abeta antibodies in two mouse models of Alzheimer diseaseStina M Fangmark Tucker, David R Borchelt, Juan C Troncoso
Plos One|January 7, 2014
An analysis of interactions between fluorescently-tagged mutant and wild-type SOD1 in intracellular inclusionsDavid A Qualls, Keith Crosby, Hilda Brown, et al.
Neurobiology of Aging|June 20, 2015
Behavioral abnormalities in APPSwe/PS1dE9 mouse model of AD-like pathology: comparative analysis across multiple behavioral domainsChristopher Janus, Abigail Y Flores, Guilian Xu, et al.
Human Molecular Genetics|September 28, 2010
An examination of wild-type SOD1 in modulating the toxicity and aggregation of ALS-associated mutant SOD1Mercedes Prudencio, Armando Durazo, Julian P Whitelegge, et al.
Nature Neuroscience|June 27, 2002
Genetically engineered mouse models of neurodegenerative diseasesPhilip C Wong, Huaibin Cai, David R Borchelt, et al.
Alzheimer'S Research & Therapy|April 28, 2012
Reduction of low-density lipoprotein receptor-related protein (LRP1) in hippocampal neurons does not proportionately reduce, or otherwise alter, amyloid deposition in APPswe/PS1dE9 transgenic miceGuilian Xu, Cameron C Green, Susan E Fromholt, et al.
Human Molecular Genetics|June 2, 2009
Variation in aggregation propensities among ALS-associated variants of SOD1: correlation to human diseaseMercedes Prudencio, P John Hart, David R Borchelt, et al.
Pageof 16