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David R Borchelt

Showing results (31-40 of 159) with videos related to

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Human Molecular Genetics|February 11, 2011
Premature death and neurologic abnormalities in transgenic mice expressing a mutant huntingtin exon-2 fragmentAndrew T N Tebbenkamp, Debbie Swing, Lino Tessarollo, et al.
Alzheimer'S Research & Therapy|September 6, 2016
Generation of a new transgenic mouse model for assessment of tau gene silencing therapiesSusan Fromholt, Christian Reitano, Hilda Brown, et al.
Molecular Neurodegeneration|January 25, 2022
Blood-based biomarkers of inflammation in amyotrophic lateral sclerosisKim A Staats, David R Borchelt, Malú Gámez Tansey, et al.
The Journal of Clinical Investigation|May 3, 2013
Thinking laterally about neurodegenerative proteinopathiesTodd E Golde, David R Borchelt, Benoit I Giasson, et al.
Translational Research : the Journal of Laboratory and Clinical Medicine|November 30, 2013
Regenerative medicine in Alzheimer's diseaseKevin M Felsenstein, Kate M Candelario, Dennis A Steindler, et al.
Plos One|November 3, 2012
Role of disulfide cross-linking of mutant SOD1 in the formation of inclusion-body-like structuresBrittany L T Roberts, Kinaree Patel, Hilda H Brown, et al.
Journal of Neuropathology and Experimental Neurology|December 25, 2003
Environmental enrichment exacerbates amyloid plaque formation in a transgenic mouse model of Alzheimer diseaseJoanna L Jankowsky, Guilian Xu, David Fromholt, et al.
Neurobiology of Aging|February 24, 2007
Differential regulation of small heat shock proteins in transgenic mouse models of neurodegenerative diseasesJiou Wang, Elizabeth Martin, Victoria Gonzales, et al.
Neuromolecular Medicine|July 14, 2007
Investigation of RNA interference to suppress expression of full-length and fragment human huntingtinDevin S Gary, Abigail Davidson, Olivier Milhavet, et al.
Human Molecular Genetics|March 21, 2013
Cytosolic proteins lose solubility as amyloid deposits in a transgenic mouse model of Alzheimer-type amyloidosisGuilian Xu, Stanley M Stevens, Brenda D Moore, et al.
Pageof 16

Showing results (31-40 of 159) with videos related to

Sort By:
Pageof 16
Human Molecular Genetics|February 11, 2011
Premature death and neurologic abnormalities in transgenic mice expressing a mutant huntingtin exon-2 fragmentAndrew T N Tebbenkamp, Debbie Swing, Lino Tessarollo, et al.
Alzheimer'S Research & Therapy|September 6, 2016
Generation of a new transgenic mouse model for assessment of tau gene silencing therapiesSusan Fromholt, Christian Reitano, Hilda Brown, et al.
Molecular Neurodegeneration|January 25, 2022
Blood-based biomarkers of inflammation in amyotrophic lateral sclerosisKim A Staats, David R Borchelt, Malú Gámez Tansey, et al.
The Journal of Clinical Investigation|May 3, 2013
Thinking laterally about neurodegenerative proteinopathiesTodd E Golde, David R Borchelt, Benoit I Giasson, et al.
Translational Research : the Journal of Laboratory and Clinical Medicine|November 30, 2013
Regenerative medicine in Alzheimer's diseaseKevin M Felsenstein, Kate M Candelario, Dennis A Steindler, et al.
Plos One|November 3, 2012
Role of disulfide cross-linking of mutant SOD1 in the formation of inclusion-body-like structuresBrittany L T Roberts, Kinaree Patel, Hilda H Brown, et al.
Journal of Neuropathology and Experimental Neurology|December 25, 2003
Environmental enrichment exacerbates amyloid plaque formation in a transgenic mouse model of Alzheimer diseaseJoanna L Jankowsky, Guilian Xu, David Fromholt, et al.
Neurobiology of Aging|February 24, 2007
Differential regulation of small heat shock proteins in transgenic mouse models of neurodegenerative diseasesJiou Wang, Elizabeth Martin, Victoria Gonzales, et al.
Neuromolecular Medicine|July 14, 2007
Investigation of RNA interference to suppress expression of full-length and fragment human huntingtinDevin S Gary, Abigail Davidson, Olivier Milhavet, et al.
Human Molecular Genetics|March 21, 2013
Cytosolic proteins lose solubility as amyloid deposits in a transgenic mouse model of Alzheimer-type amyloidosisGuilian Xu, Stanley M Stevens, Brenda D Moore, et al.
Pageof 16