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Plos One|December 24, 2010
Three novel downstream promoter elements regulate MHC class I promoter activity in mammalian cellsNamhoon Lee, Shankar S Iyer, Jie Mu, et al.
The Journal of Biological Chemistry|October 28, 2015
Kinetically Defined Mechanisms and Positions of Action of Two New Modulators of Glucocorticoid Receptor-regulated Gene InductionMadhumita A Pradhan, John A Blackford, Ballachanda N Devaiah, et al.
Endocrinology|February 28, 2002
High glucose levels increase major histocompatibility complex class I gene expression in thyroid cells and amplify interferon-gamma actionGiorgio Napolitano, Ines Bucci, Cesidio Giuliani, et al.
Cell Reports|November 21, 2019
The Bromodomain Protein 4 Contributes to the Regulation of Alternative SplicingSheetal Uppal, Anne Gegonne, Qingrong Chen, et al.
Nature Structural & Molecular Biology|May 10, 2016
BRD4 is a histone acetyltransferase that evicts nucleosomes from chromatinBallachanda N Devaiah, Chanelle Case-Borden, Anne Gegonne, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 3, 2020
MYC protein stability is negatively regulated by BRD4Ballachanda N Devaiah, Jie Mu, Ben Akman, et al.
Biorxiv : the Preprint Server for Biology|February 14, 2024
The ESCRT protein CHMP5 promotes T cell leukemia by controlling BRD4-p300-dependent transcriptionKatharine Umphred-Wilson, Shashikala Ratnayake, Qianzi Tang, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 18, 2012
BRD4 is an atypical kinase that phosphorylates serine2 of the RNA polymerase II carboxy-terminal domainBallachanda N Devaiah, Brian A Lewis, Natasha Cherman, et al.
Endocrinology|July 3, 2004
The 90K protein increases major histocompatibility complex class I expression and is regulated by hormones, gamma-interferon, and double-strand polynucleotidesAntonino Grassadonia, Nicola Tinari, Bruno Fiorentino, et al.
Immunity|June 11, 2013
Foxp3 transcription factor is proapoptotic and lethal to developing regulatory T cells unless counterbalanced by cytokine survival signalsXuguang Tai, Batu Erman, Amala Alag, et al.
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