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Genetic Epidemiology|November 16, 2004
SNPs, haplotypes, and model selection in a candidate gene region: the SIMPle analysis for multilocus dataDavid V Conti, W James Gauderman
Genetic Epidemiology|April 6, 2007
Testing association between disease and multiple SNPs in a candidate geneW James Gauderman, Cassandra Murcray, Frank Gilliland, et al.
Genetic Epidemiology|August 27, 2005
Testing association and linkage using affected-sib-parent study designsJoshua Millstein, Kimberly D Siegmund, David V Conti, et al.
American Journal of Human Genetics|December 31, 2005
A testing framework for identifying susceptibility genes in the presence of epistasisJoshua Millstein, David V Conti, Frank D Gilliland, et al.
BMC Genetics|February 3, 2006
Identifying susceptibility genes by using joint tests of association and linkage and accounting for epistasisJoshua Millstein, Kimberly D Siegmund, David V Conti, et al.
Statistics in Medicine|August 5, 2010
Discovery of complex pathways from observational dataJames W Baurley, David V Conti, W James Gauderman, et al.
American Journal of Epidemiology|January 22, 2013
Confounding and heterogeneity in genetic association studies with admixed populationsJinghua Liu, Juan Pablo Lewinger, Frank D Gilliland, et al.
Genetic Epidemiology|February 11, 2011
Sample size requirements to detect gene-environment interactions in genome-wide association studiesCassandra E Murcray, Juan Pablo Lewinger, David V Conti, et al.
Environmental Health Perspectives|March 13, 2008
Glutathione S-transferase P1, maternal smoking, and asthma in children: a haplotype-based analysisYu-Fen Li, W James Gauderman, David V Conti, et al.
American Journal of Epidemiology|January 17, 2019
A Unified Model for the Analysis of Gene-Environment InteractionW James Gauderman, Andre Kim, David V Conti, et al.
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