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Proceedings of the National Academy of Sciences of the United States of America|May 24, 2012
Knockout of the prion protein (PrP)-like Sprn gene does not produce embryonic lethality in combination with PrP(C)-deficiencyNathalie Daude, Serene Wohlgemuth, Rebecca Brown, et al.The Journal of Biological Chemistry|August 24, 2006
Cytosolic proteins regulate alpha-synuclein dissociation from presynaptic membranesSabine Wislet-Gendebien, Cheryl D'Souza, Toshitaka Kawarai, et al.Journal of Toxicology and Environmental Health. Part A|January 11, 2011
PrioNet Canada: a network of centres of excellence for research on prion diseases--ongoing and future research directionsMichelle Wong, Janie Toth, Sandra Haney, et al.Neuropsychology|December 2, 2014
ApoE and pulse pressure interactively influence level and change in the aging of episodic memory: Protective effects among ε2 carriersG Peggy McFall, Sandra A Wiebe, David Vergote, et al.Alzheimer'S & Dementia (New York, N. Y.)|October 26, 2017
Cyclic AC253, a novel amylin receptor antagonist, improves cognitive deficits in a mouse model of Alzheimer's diseaseRania Soudy, Aarti Patel, Wen Fu, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 19, 2010
Group II metabotropic glutamate receptor stimulation triggers production and release of Alzheimer's amyloid(beta)42 from isolated intact nerve terminalsSoong Ho Kim, Paul E Fraser, David Westaway, et al.Journal of Neuroinflammation|October 8, 2017
Role of microglial amylin receptors in mediating beta amyloid (Aβ)-induced inflammationWen Fu, Vlatka Vukojevic, Aarti Patel, et al.Brain Research|April 22, 2006
Loss of nicastrin elicits an apoptotic phenotype in mouse embryosVan Nguyen, Cynthia Hawkins, Catherine Bergeron, et al.Molecular Biology of the Cell|January 15, 2016
Inhibition of the FKBP family of peptidyl prolyl isomerases induces abortive translocation and degradation of the cellular prion proteinPawel Stocki, Maxime Sawicki, Charles E Mays, et al.Journal of Virology|October 2, 2015
Prion Infectivity Plateaus and Conversion to Symptomatic Disease Originate from Falling Precursor Levels and Increased Levels of Oligomeric PrPSc SpeciesCharles E Mays, Jacques van der Merwe, Chae Kim, et al.Pageof 14