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The Journal of General Physiology|November 1, 2024
Myosin-binding protein-H: Not just fillerDavid Y BarefieldThe Journal of General Physiology|March 22, 2023
Is haploinsufficiency a sufficient mechanism for MYBPC3 truncating mutations?David Y BarefieldCell Metabolism|February 5, 2015
The genetic landscape of cardiomyopathy and its role in heart failureElizabeth M McNally, David Y Barefield, Megan J PuckelwartzCurrent Cardiology Reports|April 15, 2023
Mechanisms of Sarcomere Protein Mutation-Induced CardiomyopathiesDavid Y Barefield, Alejandro Alvarez-Arce, Kelly N AraujoAmerican Journal of Physiology. Heart and Circulatory Physiology|December 29, 2023
Fine tuning contractility: atrial sarcomere function in health and diseaseHope V Burnham, Hannah E Cizauskas, David Y BarefieldJournal of Molecular Biomarkers & Diagnosis|December 20, 2016
High-Throughput Diagnostic Assay for a Highly Prevalent Cardiomyopathy-Associated MYBPC3 VariantDavid Y Barefield, Thomas L Lynch, Aravindakshan Jagadeesan, et al.The Journal of Cell Biology|June 15, 2016
An actin-dependent annexin complex mediates plasma membrane repair in muscleAlexis R Demonbreun, Mattia Quattrocelli, David Y Barefield, et al.Scientific Reports|August 6, 2021
Loss of dysferlin or myoferlin results in differential defects in excitation-contraction coupling in mouse skeletal muscleDavid Y Barefield, Jordan J Sell, Ibrahim Tahtah, et al.The Journal of Clinical Investigation|February 10, 2022
Intermittent glucocorticoid treatment enhances skeletal muscle performance through sexually dimorphic mechanismsIsabella M Salamone, Mattia Quattrocelli, David Y Barefield, et al.Heliyon|July 13, 2018
Transgenic overexpression of the SUR2A-55 splice variant in mouse heart reduces infract size and promotes protective mitochondrial functionMohun Ramratnam, Barrett Kenny, John W Kyle, et al.Pageof 4