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The Journal of Biological Chemistry|November 19, 2003
Nutrient-dependent and insulin-stimulated phosphorylation of insulin receptor substrate-1 on serine 302 correlates with increased insulin signalingJodel Giraud, Rebecca Leshan, Yong-Hee Lee, et al.
The Journal of Biological Chemistry|November 19, 2016
G protein-coupled receptors (GPCRs) That Signal via Protein Kinase A (PKA) Cross-talk at Insulin Receptor Substrate 1 (IRS1) to Activate the phosphatidylinositol 3-kinase (PI3K)/AKT PathwayNathan C Law, Morris F White, Mary E Hunzicker-Dunn
The Journal of Biological Chemistry|December 23, 2016
Down-regulation of Insulin Receptor Substrate 1 during Hyperglycemia Induces Vascular Smooth Muscle Cell DedifferentiationGang Xi, Christine Wai, Morris F White, et al.
The Biochemical Journal|August 30, 2011
Connective tissue growth factor antagonizes transforming growth factor-β1/Smad signalling in renal mesangial cellsHelen C O'Donovan, Fionnuala Hickey, Derek P Brazil, et al.
The Journal of Biological Chemistry|November 6, 2002
c-Jun N-terminal kinase (JNK) mediates feedback inhibition of the insulin signaling cascadeYong Hee Lee, Jodel Giraud, Roger J Davis, et al.
Molecular Metabolism|February 26, 2014
Irs2 and Irs4 synergize in non-LepRb neurons to control energy balance and glucose homeostasisMarianna Sadagurski, X Charlie Dong, Martin G Myers, et al.
Molecular and Cellular Biology|December 8, 2010
Insulin receptor substrates Irs1 and Irs2 coordinate skeletal muscle growth and metabolism via the Akt and AMPK pathwaysYun Chau Long, Zhiyong Cheng, Kyle D Copps, et al.
The Journal of Biological Chemistry|February 6, 2016
Serine 302 Phosphorylation of Mouse Insulin Receptor Substrate 1 (IRS1) Is Dispensable for Normal Insulin Signaling and Feedback Regulation by Hepatic S6 KinaseKyle D Copps, Nancy J Hançer, Wei Qiu, et al.
The Journal of Biological Chemistry|September 14, 2002
SOCS-1 and SOCS-3 block insulin signaling by ubiquitin-mediated degradation of IRS1 and IRS2Liangyou Rui, Minsheng Yuan, Daniel Frantz, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|November 16, 2013
Cyclic adenosine monophosphate-response element-binding protein mediates the proangiogenic or proinflammatory activity of gremlinMichela Corsini, Emanuela Moroni, Cosetta Ravelli, et al.
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