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Hypertension (Dallas, Tex. : 1979)|January 26, 2011
Angiotensinergic signaling in the brain mediates metabolic effects of deoxycorticosterone (DOCA)-salt in C57 miceJustin L Grobe, Beth A Buehrer, Aline M Hilzendeger, et al.
Hypertension (Dallas, Tex. : 1979)|December 26, 2012
Angiotensin type 1a receptors in the subfornical organ are required for deoxycorticosterone acetate-salt hypertensionAline M Hilzendeger, Martin D Cassell, Deborah R Davis, et al.
JCI Insight|November 25, 2016
Cullin-3 mutation causes arterial stiffness and hypertension through a vascular smooth muscle mechanismLarry N Agbor, Stella-Rita C Ibeawuchi, Chunyan Hu, et al.
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|July 15, 2011
Renal proximal tubule angiotensin AT1A receptors regulate blood pressureHuiping Li, Eric T Weatherford, Deborah R Davis, et al.
Hypertension (Dallas, Tex. : 1979)|October 19, 2016
Selective Deletion of the Brain-Specific Isoform of Renin Causes Neurogenic HypertensionKeisuke Shinohara, Xuebo Liu, Donald A Morgan, et al.
Physiological Genomics|December 30, 2010
Neuron- or glial-specific ablation of secreted renin does not affect renal renin, baseline arterial pressure, or metabolismDi Xu, Giulianna R Borges, Deborah R Davis, et al.
JCI Insight|March 30, 2017
Retinol-binding protein 7 is an endothelium-specific PPARγ cofactor mediating an antioxidant response through adiponectinChunyan Hu, Henry L Keen, Ko-Ting Lu, et al.
JCI Insight|June 12, 2019
Conditional deletion of smooth muscle Cullin-3 causes severe progressive hypertensionLarry N Agbor, Anand R Nair, Jing Wu, et al.
Hypertension (Dallas, Tex. : 1979)|November 25, 2015
Protective Role for Tissue Inhibitor of Metalloproteinase-4, a Novel Peroxisome Proliferator-Activated Receptor-γ Target Gene, in Smooth Muscle in Deoxycorticosterone Acetate-Salt HypertensionPimonrat Ketsawatsomkron, Henry L Keen, Deborah R Davis, et al.
Physiological Reports|January 21, 2015
Pregnant mice lacking indoleamine 2,3-dioxygenase exhibit preeclampsia phenotypesMark K Santillan, Christopher J Pelham, Pimonrat Ketsawatsomkron, et al.
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