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Decrock

Showing results (61-70 of 77) with videos related to

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Frontiers in Physiology|January 24, 2014
Connexin and pannexin (hemi)channels in the liverMichaël Maes, Elke Decrock, Bruno Cogliati, et al.
European Journal of Vascular and Endovascular Surgery : the Official Journal of the European Society for Vascular Surgery|January 29, 2013
Inhibiting connexin channels protects against cryopreservation-induced cell death in human blood vesselsM Bol, C Van Geyt, S Baert, et al.
Scientific Reports|November 14, 2019
Blocking connexin43 hemichannels protects mice against tumour necrosis factor-induced inflammatory shockTinneke Delvaeye, Maarten A J De Smet, Stijn Verwaerde, et al.
The Journal of Biological Chemistry|February 15, 2015
The BH4 domain of anti-apoptotic Bcl-XL, but not that of the related Bcl-2, limits the voltage-dependent anion channel 1 (VDAC1)-mediated transfer of pro-apoptotic Ca2+ signals to mitochondriaGiovanni Monaco, Elke Decrock, Nir Arbel, et al.
Cell Death and Differentiation|August 6, 2011
Selective regulation of IP3-receptor-mediated Ca2+ signaling and apoptosis by the BH4 domain of Bcl-2 versus Bcl-XlG Monaco, E Decrock, H Akl, et al.
Cell Calcium|August 7, 2009
Ca(2+) regulation of connexin 43 hemichannels in C6 glioma and glial cellsElke De Vuyst, Nan Wang, Elke Decrock, et al.
Toxicology Letters|July 9, 2017
Connexin hemichannel inhibition reduces acetaminophen-induced liver injury in miceMichaël Maes, Sara Crespo Yanguas, Joost Willebrords, et al.
Scientific Reports|April 16, 2015
Ryanodine receptors are targeted by anti-apoptotic Bcl-XL involving its BH4 domain and Lys87 from its BH3 domainTim Vervliet, Irma Lemmens, Elien Vandermarliere, et al.
Plos One|October 19, 2013
Alpha-helical destabilization of the Bcl-2-BH4-domain peptide abolishes its ability to inhibit the IP3 receptorGiovanni Monaco, Elke Decrock, Koen Nuyts, et al.
Cell Death and Differentiation|November 26, 2011
Transfer of IP₃ through gap junctions is critical, but not sufficient, for the spread of apoptosisE Decrock, D V Krysko, M Vinken, et al.
Pageof 8

Showing results (61-70 of 77) with videos related to

Sort By:
Pageof 8
Frontiers in Physiology|January 24, 2014
Connexin and pannexin (hemi)channels in the liverMichaël Maes, Elke Decrock, Bruno Cogliati, et al.
European Journal of Vascular and Endovascular Surgery : the Official Journal of the European Society for Vascular Surgery|January 29, 2013
Inhibiting connexin channels protects against cryopreservation-induced cell death in human blood vesselsM Bol, C Van Geyt, S Baert, et al.
Scientific Reports|November 14, 2019
Blocking connexin43 hemichannels protects mice against tumour necrosis factor-induced inflammatory shockTinneke Delvaeye, Maarten A J De Smet, Stijn Verwaerde, et al.
The Journal of Biological Chemistry|February 15, 2015
The BH4 domain of anti-apoptotic Bcl-XL, but not that of the related Bcl-2, limits the voltage-dependent anion channel 1 (VDAC1)-mediated transfer of pro-apoptotic Ca2+ signals to mitochondriaGiovanni Monaco, Elke Decrock, Nir Arbel, et al.
Cell Death and Differentiation|August 6, 2011
Selective regulation of IP3-receptor-mediated Ca2+ signaling and apoptosis by the BH4 domain of Bcl-2 versus Bcl-XlG Monaco, E Decrock, H Akl, et al.
Cell Calcium|August 7, 2009
Ca(2+) regulation of connexin 43 hemichannels in C6 glioma and glial cellsElke De Vuyst, Nan Wang, Elke Decrock, et al.
Toxicology Letters|July 9, 2017
Connexin hemichannel inhibition reduces acetaminophen-induced liver injury in miceMichaël Maes, Sara Crespo Yanguas, Joost Willebrords, et al.
Scientific Reports|April 16, 2015
Ryanodine receptors are targeted by anti-apoptotic Bcl-XL involving its BH4 domain and Lys87 from its BH3 domainTim Vervliet, Irma Lemmens, Elien Vandermarliere, et al.
Plos One|October 19, 2013
Alpha-helical destabilization of the Bcl-2-BH4-domain peptide abolishes its ability to inhibit the IP3 receptorGiovanni Monaco, Elke Decrock, Koen Nuyts, et al.
Cell Death and Differentiation|November 26, 2011
Transfer of IP₃ through gap junctions is critical, but not sufficient, for the spread of apoptosisE Decrock, D V Krysko, M Vinken, et al.
Pageof 8