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Biorxiv : the Preprint Server for Biology|July 9, 2025
The pathogenic E139D mutation stabilizes a non-canonical active state of the multi-domain phosphatase SHP2Anne E van Vlimmeren, Ziyuan Jiang, Deepti Karandur, et al.Protein Science : a Publication of the Protein Society|November 13, 2025
The pathogenic E139D mutation stabilizes a non-canonical active state of the multi-domain phosphatase SHP2Anne E van Vlimmeren, Ziyuan Jiang, Deepti Karandur, et al.Elife|June 11, 2020
CRISPR-Cas12a exploits R-loop asymmetry to form double-strand breaksJoshua C Cofsky, Deepti Karandur, Carolyn J Huang, et al.Science (New York, N.Y.)|October 12, 2019
Cryo-EM structure of a dimeric B-Raf:14-3-3 complex reveals asymmetry in the active sites of B-Raf kinasesYasushi Kondo, Jana Ognjenović, Saikat Banerjee, et al.Proceedings of the National Academy of Sciences of the United States of America|July 16, 2024
The pathogenic T42A mutation in SHP2 rewires the interaction specificity of its N-terminal regulatory domainAnne E van Vlimmeren, Rashmi Voleti, Cassandra A Chartier, et al.Biorxiv : the Preprint Server for Biology|July 28, 2023
The pathogenic T42A mutation in SHP2 rewires the interaction specificity of its N-terminal regulatory domainAnne E van Vlimmeren, Rashmi Voleti, Cassandra A Chartier, et al.Elife|March 29, 2016
Molecular basis for multimerization in the activation of the epidermal growth factor receptorYongjian Huang, Shashank Bharill, Deepti Karandur, et al.Elife|October 5, 2016
An electrostatic selection mechanism controls sequential kinase signaling downstream of the T cell receptorNeel H Shah, Qi Wang, Qingrong Yan, et al.Elife|September 9, 2020
Breakage of the oligomeric CaMKII hub by the regulatory segment of the kinaseDeepti Karandur, Moitrayee Bhattacharyya, Zijie Xia, et al.Elife|March 11, 2022
A saturation-mutagenesis analysis of the interplay between stability and activation in RasFrank Hidalgo, Laura M Nocka, Neel H Shah, et al.Pageof 2