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Diana C Parrish

Showing results (1-10 of 11) with videos related to

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Molecular and Cellular Neurosciences|January 19, 2011
Cytokines inhibit norepinephrine transporter expression by decreasing Hand2Michael J Pellegrino, Diana C Parrish, Richard E Zigmond, et al.
Neuropeptides|November 2, 2010
Cardiac ischemia-reperfusion regulates sympathetic neuropeptide expression through gp130-dependent and independent mechanismsEric N Alston, Diana C Parrish, Wohaib Hasan, et al.
Elife|May 23, 2022
Chondroitin sulfate proteoglycan 4,6 sulfation regulates sympathetic nerve regeneration after myocardial infarctionMatthew R Blake, Diana C Parrish, Melanie A Staffenson, et al.
American Journal of Physiology. Heart and Circulatory Physiology|October 24, 2007
Postinfarct sympathetic hyperactivity differentially stimulates expression of tyrosine hydroxylase and norepinephrine transporterDiana C Parrish, Kurt Gritman, Donna M Van Winkle, et al.
Experimental Physiology|November 3, 2009
Infarction-induced cytokines cause local depletion of tyrosine hydroxylase in cardiac sympathetic nervesDiana C Parrish, Eric N Alston, Hermann Rohrer, et al.
Physiological Reports|May 25, 2023
Loss of chondroitin sulfate proteoglycan sulfation allows delayed sympathetic reinnervation after cardiac ischemia-reperfusionMatthew R Blake, Diana C Parrish, Melanie A Staffenson, et al.
American Journal of Physiology. Heart and Circulatory Physiology|November 5, 2017
Transient denervation of viable myocardium after myocardial infarction does not alter arrhythmia susceptibilityDiana C Parrish, Samantha D Francis Stuart, Antoinette Olivas, et al.
Experimental Neurology|September 10, 2013
Sympathetic denervation of peri-infarct myocardium requires the p75 neurotrophin receptorChristina U Lorentz, Diana C Parrish, Eric N Alston, et al.
American Journal of Physiology. Heart and Circulatory Physiology|July 14, 2009
Absence of gp130 in dopamine beta-hydroxylase-expressing neurons leads to autonomic imbalance and increased reperfusion arrhythmiasDiana C Parrish, Eric N Alston, Hermann Rohrer, et al.
The Journal of Experimental Medicine|October 24, 2012
ProNGF, a cytokine induced after myocardial infarction in humans, targets pericytes to promote microvascular damage and activationChia-Jen Siao, Christina U Lorentz, Pouneh Kermani, et al.
Pageof 2

Showing results (1-10 of 11) with videos related to

Sort By:
Pageof 2
Molecular and Cellular Neurosciences|January 19, 2011
Cytokines inhibit norepinephrine transporter expression by decreasing Hand2Michael J Pellegrino, Diana C Parrish, Richard E Zigmond, et al.
Neuropeptides|November 2, 2010
Cardiac ischemia-reperfusion regulates sympathetic neuropeptide expression through gp130-dependent and independent mechanismsEric N Alston, Diana C Parrish, Wohaib Hasan, et al.
Elife|May 23, 2022
Chondroitin sulfate proteoglycan 4,6 sulfation regulates sympathetic nerve regeneration after myocardial infarctionMatthew R Blake, Diana C Parrish, Melanie A Staffenson, et al.
American Journal of Physiology. Heart and Circulatory Physiology|October 24, 2007
Postinfarct sympathetic hyperactivity differentially stimulates expression of tyrosine hydroxylase and norepinephrine transporterDiana C Parrish, Kurt Gritman, Donna M Van Winkle, et al.
Experimental Physiology|November 3, 2009
Infarction-induced cytokines cause local depletion of tyrosine hydroxylase in cardiac sympathetic nervesDiana C Parrish, Eric N Alston, Hermann Rohrer, et al.
Physiological Reports|May 25, 2023
Loss of chondroitin sulfate proteoglycan sulfation allows delayed sympathetic reinnervation after cardiac ischemia-reperfusionMatthew R Blake, Diana C Parrish, Melanie A Staffenson, et al.
American Journal of Physiology. Heart and Circulatory Physiology|November 5, 2017
Transient denervation of viable myocardium after myocardial infarction does not alter arrhythmia susceptibilityDiana C Parrish, Samantha D Francis Stuart, Antoinette Olivas, et al.
Experimental Neurology|September 10, 2013
Sympathetic denervation of peri-infarct myocardium requires the p75 neurotrophin receptorChristina U Lorentz, Diana C Parrish, Eric N Alston, et al.
American Journal of Physiology. Heart and Circulatory Physiology|July 14, 2009
Absence of gp130 in dopamine beta-hydroxylase-expressing neurons leads to autonomic imbalance and increased reperfusion arrhythmiasDiana C Parrish, Eric N Alston, Hermann Rohrer, et al.
The Journal of Experimental Medicine|October 24, 2012
ProNGF, a cytokine induced after myocardial infarction in humans, targets pericytes to promote microvascular damage and activationChia-Jen Siao, Christina U Lorentz, Pouneh Kermani, et al.
Pageof 2