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Diego Sepulveda-Falla

Showing results (11-20 of 53) with videos related to

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Cells|October 14, 2022
Loss of Homeostatic Microglia Signature in Prion DiseasesYue Wang, Kristin Hartmann, Edda Thies, et al.
Frontiers in Aging Neuroscience|August 28, 2020
Decreased Deposition of Beta-Amyloid 1-38 and Increased Deposition of Beta-Amyloid 1-42 in Brain Tissue of Presenilin-1 E280A Familial Alzheimer's Disease PatientsFelix Dinkel, Diana Trujillo-Rodriguez, Andres Villegas, et al.
Brain : a Journal of Neurology|February 13, 2014
High molecular mass assemblies of amyloid-β oligomers bind prion protein in patients with Alzheimer's diseaseFrank Dohler, Diego Sepulveda-Falla, Susanne Krasemann, et al.
Neuropathology : Official Journal of the Japanese Society of Neuropathology|October 16, 2018
Distinct microglia profile in Creutzfeldt-Jakob disease and Alzheimer's disease is independent of disease kineticsKatarina Krbot, Peter Hermann, Magdalena Krbot Skorić, et al.
Neuroreport|October 4, 2013
Specific de-SUMOylation triggered by acquisition of spatial learning is related to epigenetic changes in the rat hippocampusSergio Castro-Gomez, Alvaro Barrera-Ocampo, Gloria Machado-Rodriguez, et al.
Neuropathology : Official Journal of the Japanese Society of Neuropathology|November 18, 2025
ApoE4 Homozygosity Is Associated With Increased Microglia Activation in Fatal COVID-19Amar Hamdan, Yassir El-Amri, Fabian Heinrich, et al.
Journal of Neuropathology and Experimental Neurology|August 4, 2016
Amyloid-β Precursor Protein Modulates the Sorting of Testican-1 and Contributes to Its Accumulation in Brain Tissue and Cerebrospinal Fluid from Patients with Alzheimer DiseaseAlvaro Barrera-Ocampo, Sönke Arlt, Jakob Matschke, et al.
Acta Neuropathologica Communications|May 24, 2019
Complement 3<sup>+</sup>-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microgliaKristin Hartmann, Diego Sepulveda-Falla, Indigo V L Rose, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|March 7, 2024
Genetic modifiers of cognitive decline in PSEN1 E280A Alzheimer's diseaseDiego Sepulveda-Falla, Jorge I Vélez, Natalia Acosta-Baena, et al.
The Lancet. Neurology|February 8, 2011
Pre-dementia clinical stages in presenilin 1 E280A familial early-onset Alzheimer's disease: a retrospective cohort studyNatalia Acosta-Baena, Diego Sepulveda-Falla, Carlos Mario Lopera-Gómez, et al.
Pageof 6

Showing results (11-20 of 53) with videos related to

Sort By:
Pageof 6
Cells|October 14, 2022
Loss of Homeostatic Microglia Signature in Prion DiseasesYue Wang, Kristin Hartmann, Edda Thies, et al.
Frontiers in Aging Neuroscience|August 28, 2020
Decreased Deposition of Beta-Amyloid 1-38 and Increased Deposition of Beta-Amyloid 1-42 in Brain Tissue of Presenilin-1 E280A Familial Alzheimer's Disease PatientsFelix Dinkel, Diana Trujillo-Rodriguez, Andres Villegas, et al.
Brain : a Journal of Neurology|February 13, 2014
High molecular mass assemblies of amyloid-β oligomers bind prion protein in patients with Alzheimer's diseaseFrank Dohler, Diego Sepulveda-Falla, Susanne Krasemann, et al.
Neuropathology : Official Journal of the Japanese Society of Neuropathology|October 16, 2018
Distinct microglia profile in Creutzfeldt-Jakob disease and Alzheimer's disease is independent of disease kineticsKatarina Krbot, Peter Hermann, Magdalena Krbot Skorić, et al.
Neuroreport|October 4, 2013
Specific de-SUMOylation triggered by acquisition of spatial learning is related to epigenetic changes in the rat hippocampusSergio Castro-Gomez, Alvaro Barrera-Ocampo, Gloria Machado-Rodriguez, et al.
Neuropathology : Official Journal of the Japanese Society of Neuropathology|November 18, 2025
ApoE4 Homozygosity Is Associated With Increased Microglia Activation in Fatal COVID-19Amar Hamdan, Yassir El-Amri, Fabian Heinrich, et al.
Journal of Neuropathology and Experimental Neurology|August 4, 2016
Amyloid-β Precursor Protein Modulates the Sorting of Testican-1 and Contributes to Its Accumulation in Brain Tissue and Cerebrospinal Fluid from Patients with Alzheimer DiseaseAlvaro Barrera-Ocampo, Sönke Arlt, Jakob Matschke, et al.
Acta Neuropathologica Communications|May 24, 2019
Complement 3<sup>+</sup>-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microgliaKristin Hartmann, Diego Sepulveda-Falla, Indigo V L Rose, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|March 7, 2024
Genetic modifiers of cognitive decline in PSEN1 E280A Alzheimer's diseaseDiego Sepulveda-Falla, Jorge I Vélez, Natalia Acosta-Baena, et al.
The Lancet. Neurology|February 8, 2011
Pre-dementia clinical stages in presenilin 1 E280A familial early-onset Alzheimer's disease: a retrospective cohort studyNatalia Acosta-Baena, Diego Sepulveda-Falla, Carlos Mario Lopera-Gómez, et al.
Pageof 6