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Trends in Endocrinology and Metabolism: TEM|July 4, 2015
Cardiac contraction-induced GLUT4 translocation requires dual signaling inputJoost J F P Luiken, Jan F C Glatz, Dietbert Neumann
The Journal of Biological Chemistry|September 1, 2006
Dissecting the role of 5'-AMP for allosteric stimulation, activation, and deactivation of AMP-activated protein kinaseMarianne Suter, Uwe Riek, Roland Tuerk, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|March 27, 2020
Understanding the distinct subcellular trafficking of CD36 and GLUT4 during the development of myocardial insulin resistanceJoost J F P Luiken, Miranda Nabben, Dietbert Neumann, et al.
Prostaglandins, Leukotrienes, and Essential Fatty Acids|July 4, 2016
Molecular mechanism of lipid-induced cardiac insulin resistance and contractile dysfunctionYilin Liu, Dietbert Neumann, Jan F C Glatz, et al.
FEBS Letters|December 9, 2008
Myosin light chains are not a physiological substrate of AMPK in the control of cell structure changesLaurent Bultot, Sandrine Horman, Dietbert Neumann, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 21, 2010
The PP1-R6 protein phosphatase holoenzyme is involved in the glucose-induced dephosphorylation and inactivation of AMP-activated protein kinase, a key regulator of insulin secretion, in MIN6 beta cellsLuisa Garcia-Haro, Maria Adelaida Garcia-Gimeno, Dietbert Neumann, et al.
Biochimica Et Biophysica Acta|September 13, 2016
Post-translational modifications of CD36 (SR-B2): Implications for regulation of myocellular fatty acid uptakeJoost J F P Luiken, Dipanjan Chanda, Miranda Nabben, et al.
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