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Current Opinion in Neurobiology|November 12, 2015
An amyloid-like cascade hypothesis for C9orf72 ALS/FTDDieter Edbauer, Christian HaassJournal of Molecular Medicine (Berlin, Germany)|August 27, 2013
FUS-mediated alternative splicing in the nervous system: consequences for ALS and FTLDDenise Orozco, Dieter EdbauerScience (New York, N.Y.)|October 6, 2022
A perturbed network in neurodegenerationJean-Marc Gallo, Dieter EdbauerCell Communication and Signaling : CCS|April 30, 2014
Good guy or bad guy: the opposing roles of microRNA 125b in cancerJulia Banzhaf-Strathmann, Dieter EdbauerEMBO Reports|June 15, 2023
C9orf72 protein quality control by UBR5-mediated heterotypic ubiquitin chainsJulia Jülg, Dieter Edbauer, Christian BehrendsThe Journal of Biological Chemistry|June 24, 2004
Co-expression of nicastrin and presenilin rescues a loss of function mutant of APH-1Dieter Edbauer, Christoph Kaether, Harald Steiner, et al.Proceedings of the National Academy of Sciences of the United States of America|June 6, 2002
Presenilin and nicastrin regulate each other and determine amyloid beta-peptide production via complex formationDieter Edbauer, Edith Winkler, Christian Haass, et al.The Journal of Biological Chemistry|March 25, 2004
Requirement of PEN-2 for stabilization of the presenilin N-/C-terminal fragment heterodimer within the gamma-secretase complexStefan Prokop, Keiro Shirotani, Dieter Edbauer, et al.Journal of Neurochemistry|June 11, 2004
Immature nicastrin stabilizes APH-1 independent of PEN-2 and presenilin: identification of nicastrin mutants that selectively interact with APH-1Keiro Shirotani, Dieter Edbauer, Marcus Kostka, et al.Pageof 11