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EMBO Molecular Medicine|May 24, 2018
A novel CHCHD10 mutation implicates a Mia40-dependent mitochondrial import deficit in ALSCarina Lehmer, Martin H Schludi, Linnea Ransom, et al.Frontiers in Cell and Developmental Biology|June 29, 2023
Loss of TDP-43 causes ectopic endothelial sprouting and migration defects through increased fibronectin, vcam 1 and integrin α4/β1Katrin Hipke, Bettina Pitter, Alexander Hruscha, et al.The EMBO Journal|February 3, 2022
Disease-linked TDP-43 hyperphosphorylation suppresses TDP-43 condensation and aggregationLara A Gruijs da Silva, Francesca Simonetti, Saskia Hutten, et al.Molecular Neurodegeneration|June 20, 2024
Mis-localization of endogenous TDP-43 leads to ALS-like early-stage metabolic dysfunction and progressive motor deficitsYiying Hu, Alexander Hruscha, Chenchen Pan, et al.Acta Neuropathologica|August 15, 2014
C9orf72 FTLD/ALS-associated Gly-Ala dipeptide repeat proteins cause neuronal toxicity and Unc119 sequestrationStephanie May, Daniel Hornburg, Martin H Schludi, et al.Cell|February 6, 2018
In Situ Structure of Neuronal C9orf72 Poly-GA Aggregates Reveals Proteasome RecruitmentQiang Guo, Carina Lehmer, Antonio Martínez-Sánchez, et al.Cell Reports|December 28, 2020
Nuclear Import Receptors Directly Bind to Arginine-Rich Dipeptide Repeat Proteins and Suppress Their Pathological InteractionsSaskia Hutten, Sinem Usluer, Benjamin Bourgeois, et al.EMBO Reports|April 19, 2022
Gel-like inclusions of C-terminal fragments of TDP-43 sequester stalled proteasomes in neuronsHenrick Riemenschneider, Qiang Guo, Jakob Bader, et al.The EMBO Journal|March 17, 2020
Cell-to-cell transmission of C9orf72 poly-(Gly-Ala) triggers key features of ALS/FTDBahram Khosravi, Kathrine D LaClair, Henrick Riemenschneider, et al.Neurobiology of Aging|April 1, 2021
Low-degree trisomy 21 mosaicism promotes early-onset Alzheimer diseaseGeorg S Nuebling, Catharina Prix, Matthias Brendel, et al.Pageof 11