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Frontiers in Pharmacology|April 15, 2022
Targeting the Interaction of GABAB Receptors With CHOP After an Ischemic Insult Restores Receptor Expression and Inhibits Progressive Neuronal DeathMusadiq A Bhat, Abolghasem Esmaeili, Elena Neumann, et al.
Frontiers in Molecular Neuroscience|September 20, 2021
Sustained Baclofen-Induced Activation of GABA Receptors After Cerebral Ischemia Restores Receptor Expression and Function and Limits Progressing Loss of NeuronsMohammad Hleihil, Markus Vaas, Musadiq A Bhat, et al.
Scientific Reports|January 8, 2025
The E3 ubiquitin ligase MARCH1 mediates downregulation of plasma membrane GABAB receptors under ischemic conditions by inhibiting fast receptor recyclingMusadiq A Bhat, Mohammad Hleihil, Irene Mondéjar, et al.
The Journal of Biological Chemistry|September 10, 2010
Sustained glutamate receptor activation down-regulates GABAB receptors by shifting the balance from recycling to lysosomal degradationPatrick J Maier, Isabel Marin, Thomas Grampp, et al.
Journal of Neurochemistry|May 22, 2009
The alpha5(H105R) mutation impairs alpha5 selective binding properties by altered positioning of the alpha5 subunit in GABAA receptors containing two distinct types of alpha subunitsEla Balic, Uwe Rudolph, Jean-Marc Fritschy, et al.
Science Advances|June 18, 2020
Astrocyte dysfunction increases cortical dendritic excitability and promotes cranial pain in familial migraineJennifer Romanos, Dietmar Benke, Daniela Pietrobon, et al.
The Journal of Biological Chemistry|August 12, 2004
Analysis of the presence and abundance of GABAA receptors containing two different types of alpha subunits in murine brain using point-mutated alpha subunitsDietmar Benke, Panagiotis Fakitsas, Christian Roggenmoser, et al.
Communications Biology|April 10, 2019
Differences in glutamate uptake between cortical regions impact neuronal NMDA receptor activationJennifer Romanos, Dietmar Benke, Aiman S Saab, et al.
Frontiers in Cardiovascular Medicine|October 11, 2021
Adeno-Associated Virus-Mediated Gain-of-Function mPCSK9 Expression in the Mouse Induces Hypercholesterolemia, Monocytosis, Neutrophilia, and a Hypercoagulative StateGeorgios Louloudis, Samuele Ambrosini, Francesco Paneni, et al.
Frontiers in Cardiovascular Medicine|January 31, 2022
Corrigendum: Adeno-Associated Virus-Mediated Gain-of-Function mPCSK9 Expression in the Mouse Induces Hypercholesterolemia, Monocytosis, Neutrophilia, and a Hypercoagulative StateGeorgios Louloudis, Samuele Ambrosini, Francesco Paneni, et al.
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