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Biochemical and Biophysical Research Communications|April 11, 2015
Molecular imaging of inflammation in the ApoE -/- mouse model of atherosclerosis with IodoDPACatherine A Foss, Djahida Bedja, Ronnie C Mease, et al.Journal of the American College of Cardiology|January 10, 2009
Sildenafil stops progressive chamber, cellular, and molecular remodeling and improves calcium handling and function in hearts with pre-existing advanced hypertrophy caused by pressure overloadTakahiro Nagayama, Steven Hsu, Manling Zhang, et al.Journal of Molecular and Cellular Cardiology|January 23, 2009
Pressure-overload magnitude-dependence of the anti-hypertrophic efficacy of PDE5A inhibitionTakahiro Nagayama, Steven Hsu, Manling Zhang, et al.Clinical Science (London, England : 1979)|July 18, 2015
Impaired mitochondrial energy supply coupled to increased H2O2 emission under energy/redox stress leads to myocardial dysfunction during Type I diabetesCarlo G Tocchetti, Brian A Stanley, Vidhya Sivakumaran, et al.Journal of Molecular and Cellular Cardiology|February 16, 2010
Evidence for a role of immunoproteasomes in regulating cardiac muscle mass in diabetic miceLingyun Zu, Djahida Bedja, Karen Fox-Talbot, et al.Plos One|August 23, 2012
Cardiac-specific over-expression of epidermal growth factor receptor 2 (ErbB2) induces pro-survival pathways and hypertrophic cardiomyopathy in micePolina Sysa-Shah, Yi Xu, Xin Guo, et al.Circulation. Heart Failure|February 16, 2019
Chronic Atrial and Ventricular Pacing in the MouseMarcus Ståhlberg, Ryo Nakagawa, Djahida Bedja, et al.Plos One|November 22, 2008
Disruption of Nrf2, a key inducer of antioxidant defenses, attenuates ApoE-mediated atherosclerosis in miceThomas E Sussan, Jonathan Jun, Rajesh Thimmulappa, et al.Circulation Research|June 13, 2009
Avoidance of transient cardiomyopathy in cardiomyocyte-targeted tamoxifen-induced MerCreMer gene deletion modelsNorimichi Koitabashi, Djahida Bedja, Ari L Zaiman, et al.Circulation Research|January 23, 2014
Hyperactive adverse mechanical stress responses in dystrophic heart are coupled to transient receptor potential canonical 6 and blocked by cGMP-protein kinase G modulationKinya Seo, Peter P Rainer, Dong-Ik Lee, et al.Pageof 11