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Dominic M Walsh

Showing results (11-20 of 127) with videos related to

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The Lancet. Neurology|July 25, 2003
Soluble oligomers for the diagnosis of neurodegenerative diseasesOmar M A El-Agnaf, Dominic M Walsh, David Allsop
Biochemistry|October 19, 2016
The Aggregation Paths and Products of Aβ42 Dimers Are Distinct from Those of the Aβ42 MonomerTiernan T O'Malley, William M Witbold, Sara Linse, et al.
Cerebral Cortex (New York, N.Y. : 1991)|July 9, 2016
Peripheral Interventions Enhancing Brain Glutamate Homeostasis Relieve Amyloid β- and TNFα- Mediated Synaptic Plasticity Disruption in the Rat HippocampusDainan Zhang, Alexandra J Mably, Dominic M Walsh, et al.
Neuroscience Letters|August 4, 2004
Amyloid beta-protein induced electrophysiological changes are dependent on aggregation state: N-methyl-D-aspartate (NMDA) versus non-NMDA receptor/channel activationChianping Ye, Dominic M Walsh, Dennis J Selkoe, et al.
Molecular Medicine (Cambridge, Mass.)|December 14, 2011
β-Amyloid carrying the Dutch mutation has diverse effects on calpain-mediated toxicity in hippocampal neuronsAlexandra M Nicholson, Lindsey A Wold, Dominic M Walsh, et al.
Brain : a Journal of Neurology|August 6, 2008
Soluble amyloid-beta peptides potently disrupt hippocampal synaptic plasticity in the absence of cerebrovascular dysfunction in vivoNeng-Wei Hu, Imelda M Smith, Dominic M Walsh, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 6, 2017
Large Soluble Oligomers of Amyloid β-Protein from Alzheimer Brain Are Far Less Neuroactive Than the Smaller Oligomers to Which They DissociateTing Yang, Shaomin Li, Huixin Xu, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 2, 2004
Block of long-term potentiation by naturally secreted and synthetic amyloid beta-peptide in hippocampal slices is mediated via activation of the kinases c-Jun N-terminal kinase, cyclin-dependent kinase 5, and p38 mitogen-activated protein kinase as well as metabotropic glutamate receptor type 5Qinwen Wang, Dominic M Walsh, Michael J Rowan, et al.
The Journal of Physiology|February 14, 2006
Effects of secreted oligomers of amyloid beta-protein on hippocampal synaptic plasticity: a potent role for trimersMatthew Townsend, Ganesh M Shankar, Tapan Mehta, et al.
Molecular Medicine (Cambridge, Mass.)|March 28, 2008
Protein aggregation in the brain: the molecular basis for Alzheimer's and Parkinson's diseasesG Brent Irvine, Omar M El-Agnaf, Ganesh M Shankar, et al.
Pageof 13

Showing results (11-20 of 127) with videos related to

Sort By:
Pageof 13
The Lancet. Neurology|July 25, 2003
Soluble oligomers for the diagnosis of neurodegenerative diseasesOmar M A El-Agnaf, Dominic M Walsh, David Allsop
Biochemistry|October 19, 2016
The Aggregation Paths and Products of Aβ42 Dimers Are Distinct from Those of the Aβ42 MonomerTiernan T O'Malley, William M Witbold, Sara Linse, et al.
Cerebral Cortex (New York, N.Y. : 1991)|July 9, 2016
Peripheral Interventions Enhancing Brain Glutamate Homeostasis Relieve Amyloid β- and TNFα- Mediated Synaptic Plasticity Disruption in the Rat HippocampusDainan Zhang, Alexandra J Mably, Dominic M Walsh, et al.
Neuroscience Letters|August 4, 2004
Amyloid beta-protein induced electrophysiological changes are dependent on aggregation state: N-methyl-D-aspartate (NMDA) versus non-NMDA receptor/channel activationChianping Ye, Dominic M Walsh, Dennis J Selkoe, et al.
Molecular Medicine (Cambridge, Mass.)|December 14, 2011
β-Amyloid carrying the Dutch mutation has diverse effects on calpain-mediated toxicity in hippocampal neuronsAlexandra M Nicholson, Lindsey A Wold, Dominic M Walsh, et al.
Brain : a Journal of Neurology|August 6, 2008
Soluble amyloid-beta peptides potently disrupt hippocampal synaptic plasticity in the absence of cerebrovascular dysfunction in vivoNeng-Wei Hu, Imelda M Smith, Dominic M Walsh, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 6, 2017
Large Soluble Oligomers of Amyloid β-Protein from Alzheimer Brain Are Far Less Neuroactive Than the Smaller Oligomers to Which They DissociateTing Yang, Shaomin Li, Huixin Xu, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 2, 2004
Block of long-term potentiation by naturally secreted and synthetic amyloid beta-peptide in hippocampal slices is mediated via activation of the kinases c-Jun N-terminal kinase, cyclin-dependent kinase 5, and p38 mitogen-activated protein kinase as well as metabotropic glutamate receptor type 5Qinwen Wang, Dominic M Walsh, Michael J Rowan, et al.
The Journal of Physiology|February 14, 2006
Effects of secreted oligomers of amyloid beta-protein on hippocampal synaptic plasticity: a potent role for trimersMatthew Townsend, Ganesh M Shankar, Tapan Mehta, et al.
Molecular Medicine (Cambridge, Mass.)|March 28, 2008
Protein aggregation in the brain: the molecular basis for Alzheimer's and Parkinson's diseasesG Brent Irvine, Omar M El-Agnaf, Ganesh M Shankar, et al.
Pageof 13