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The Journal of Biological Chemistry|November 13, 2004
Mitochondrial release of pro-apoptotic proteins: electrostatic interactions can hold cytochrome c but not Smac/DIABLO to mitochondrial membranesRachel T Uren, Grant Dewson, Christine Bonzon, et al.
Molecular Biology of the Cell|November 21, 2014
Visual and functional demonstration of growing Bax-induced pores in mitochondrial outer membranesLaura A Gillies, Han Du, Bjoern Peters, et al.
Plos Biology|June 11, 2019
Phenotypic selection with an intrabody library reveals an anti-apoptotic function of PKM2 requiring Mitofusin-1Tong Liu, Tomomi Kuwana, Hongkai Zhang, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 26, 2006
Different mitochondrial intermembrane space proteins are released during apoptosis in a manner that is coordinately initiated but can vary in durationCristina Muñoz-Pinedo, Ana Guío-Carrión, Joshua C Goldstein, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 7, 2005
The multidomain proapoptotic molecules Bax and Bak are directly activated by heatLisa J Pagliari, Tomomi Kuwana, Christine Bonzon, et al.
Molecular Cell|February 22, 2005
BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectlyTomomi Kuwana, Lisa Bouchier-Hayes, Jerry E Chipuk, et al.
The Journal of Biological Chemistry|January 5, 2020
Mitochondrial residence of the apoptosis inducer BAX is more important than BAX oligomerization in promoting membrane permeabilizationTomomi Kuwana, Louise E King, Katia Cosentino, et al.
Science (New York, N.Y.)|February 14, 2004
Direct activation of Bax by p53 mediates mitochondrial membrane permeabilization and apoptosisJerry E Chipuk, Tomomi Kuwana, Lisa Bouchier-Hayes, et al.
Cell|November 7, 2002
Bid, Bax, and lipids cooperate to form supramolecular openings in the outer mitochondrial membraneTomomi Kuwana, Mason R Mackey, Guy Perkins, et al.
Molecular Cell|August 12, 2008
Opa1-mediated cristae opening is Bax/Bak and BH3 dependent, required for apoptosis, and independent of Bak oligomerizationRyuji Yamaguchi, Lydia Lartigue, Guy Perkins, et al.
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