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American Journal of Physiology. Renal Physiology|May 22, 2015
Nephron-specific deletion of the prorenin receptor causes a urine concentration defectNirupama Ramkumar, Deborah Stuart, Matias Calquin, et al.Hypertension (Dallas, Tex. : 1979)|April 24, 2013
Renal collecting duct NOS1 maintains fluid-electrolyte homeostasis and blood pressureKelly A Hyndman, Erika I Boesen, Ahmed A Elmarakby, et al.Kidney International|January 16, 2009
Inactivation of Pkd1 in principal cells causes a more severe cystic kidney disease than in intercalated cellsKalani L Raphael, Kevin A Strait, Peter K Stricklett, et al.Nature Medicine|July 12, 2005
Thiazolidinediones expand body fluid volume through PPARgamma stimulation of ENaC-mediated renal salt absorptionYouFei Guan, Chuanming Hao, Dae Ryong Cha, et al.Kidney International Reports|November 29, 2023
Targeting the Endothelin A Receptor in IgA NephropathyDonald E Kohan, Jonathan Barratt, Hiddo J L Heerspink, et al.The Journal of Clinical Investigation|August 18, 2004
Collecting duct-specific knockout of endothelin-1 causes hypertension and sodium retentionDowhan Ahn, Yuqiang Ge, Peter K Stricklett, et al.JCI Insight|April 5, 2019
Site-1 protease-derived soluble (pro)renin receptor targets vasopressin receptor 2 to enhance urine concentrating capabilityFei Wang, Chuanming Xu, Renfei Luo, et al.Pharmacological Reviews|March 10, 2016
EndothelinAnthony P Davenport, Kelly A Hyndman, Neeraj Dhaun, et al.American Journal of Physiology. Renal Physiology|December 20, 2021
Multiomic identification of factors associated with progression to cystic kidney disease in mice with nephron Ift88 disruptionChunyan Hu, Katherine Beebe, Edgar J Hernandez, et al.American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation|January 22, 2013
Why not nephrology? A survey of US internal medicine subspecialty fellowsKenar D Jhaveri, Matthew A Sparks, Hitesh H Shah, et al.Pageof 16