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The Journal of Clinical Investigation|July 2, 2026
Extracellular matrix reprogramming by the YAP/TAZ- TGF-ß2 axis drives immune exclusion in cholangiocarcinoma modelsMarco Jessen, KyungMok Kim, Marie Tollot-Wegner, et al.The Journal of Clinical Investigation|August 1, 2024
GPR126 is a specifier of blood-brain barrier formation in the mouse central nervous systemNikolaos Kakogiannos, Anna Agata Scalise, Emanuele Martini, et al.Cell|September 14, 2024
Intercellular nanotube-mediated mitochondrial transfer enhances T cell metabolic fitness and antitumor efficacyJeremy G Baldwin, Christoph Heuser-Loy, Tanmoy Saha, et al.Cell|June 19, 2024
Therapeutic potential of co-signaling receptor modulation in hepatitis BFrancesco Andreata, Chiara Laura, Micol Ravà, et al.Nature Immunology|June 30, 2025
CD4+ T cells license Kupffer cells to reverse CD8+ T cell dysfunction induced by hepatocellular primingValentina Venzin, Cristian G Beccaria, Chiara Perucchini, et al.Cancer Cell|September 19, 2019
Age-Related Gliosis Promotes Central Nervous System Lymphoma through CCL19-Mediated Tumor Cell RetentionTracy O'Connor, Xiaolan Zhou, Jan Kosla, et al.Nature|March 25, 2021
Auto-aggressive CXCR6+ CD8 T cells cause liver immune pathology in NASHMichael Dudek, Dominik Pfister, Sainitin Donakonda, et al.Nature Medicine|April 3, 2019
Platelet GPIbα is a mediator and potential interventional target for NASH and subsequent liver cancerMohsen Malehmir, Dominik Pfister, Suchira Gallage, et al.Journal of Hepatology|May 24, 2023
Intestinal B cells license metabolic T-cell activation in NASH microbiota/antigen-independently and contribute to fibrosis by IgA-FcR signallingElena Kotsiliti, Valentina Leone, Svenja Schuehle, et al.Nature|March 25, 2021
NASH limits anti-tumour surveillance in immunotherapy-treated HCCDominik Pfister, Nicolás Gonzalo Núñez, Roser Pinyol, et al.Pageof 3