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International Journal of Molecular Sciences|July 29, 2023
Single Cell Transcriptomic Analysis in a Mouse Model of Barth Syndrome Reveals Cell-Specific Alterations in Gene Expression and Intercellular CommunicationGayani Perera, Liam Power, Amy Larson, et al.Life Science Alliance|November 27, 2023
RNA helicase EIF4A1-mediated translation is essential for the GC responseMichael Screen, Louise S Matheson, Andrew Jm Howden, et al.Biochimica Et Biophysica Acta. Molecular and Cell Biology of Lipids|April 26, 2018
Loss of tafazzin results in decreased myoblast differentiation in C2C12 cells: A myoblast model of Barth syndrome and cardiolipin deficiencyWenjia Lou, Christian A Reynolds, Yiran Li, et al.Circulation Research|March 10, 2020
AAV Gene Therapy Prevents and Reverses Heart Failure in a Murine Knockout Model of Barth SyndromeSuya Wang, Yifei Li, Yang Xu, et al.The Journal of Investigative Dermatology|August 11, 2020
The RAC1 Target NCKAP1 Plays a Crucial Role in the Progression of Braf;Pten-Driven Melanoma in MiceKarthic Swaminathan, Andrew Campbell, Vassilis Papalazarou, et al.The Journal of Cell Biology|March 28, 2019
Extramitochondrial cardiolipin suggests a novel function of mitochondria in spermatogenesisMindong Ren, Yang Xu, Hediye Erdjument-Bromage, et al.Developmental Cell|June 4, 2013
TIGAR is required for efficient intestinal regeneration and tumorigenesisEric C Cheung, Dimitris Athineos, Pearl Lee, et al.Biology|September 28, 2023
Phenotypic Characterization of Female Carrier Mice Heterozygous for Tafazzin DeletionMichelle V Tomczewski, John Z Chan, Duaa M Al-Majmaie, et al.Journal of Lipid Research|May 9, 2025
Plaat1 deficiency reduces cardiac cardiolipin content and impairs exercise toleranceAshkan Hashemi, Ming Rong Liu, John Z Chan, et al.European Journal of Cell Biology|June 5, 2012
Tissue inducible Lifeact expression allows visualization of actin dynamics in vivo and ex vivoHannah Schachtner, Ang Li, David Stevenson, et al.Pageof 6