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Development (Cambridge, England)|July 21, 2016
Gestational stress induces the unfolded protein response, resulting in heart defectsHongjun Shi, Victoria C O'Reilly, Julie L M Moreau, et al.
Developmental Biology|March 25, 2014
Gene-environment interaction demonstrates the vulnerability of the embryonic heartVictoria C O'Reilly, Kylie Lopes Floro, Hongjun Shi, et al.
Human Molecular Genetics|October 26, 2014
Compound heterozygous mutations in RIPPLY2 associated with vertebral segmentation defectsAideen M McInerney-Leo, Duncan B Sparrow, Jessica E Harris, et al.
Frontiers in Genetics|February 7, 2022
Analysis of Placental Arteriovenous Formation Reveals New Insights Into Embryos With Congenital Heart DefectsJacinta I Kalisch-Smith, Emily C Morris, Mary A A Strevens, et al.
Genes & Development|November 5, 2008
BMP/SMAD1 signaling sets a threshold for the left/right pathway in lateral plate mesoderm and limits availability of SMAD4Milena B Furtado, Mark J Solloway, Vanessa J Jones, et al.
Cell|April 10, 2012
A mechanism for gene-environment interaction in the etiology of congenital scoliosisDuncan B Sparrow, Gavin Chapman, Allanceson J Smith, et al.
Development (Cambridge, England)|February 22, 2019
Gene-environment interaction impacts on heart development and embryo survivalJulie L M Moreau, Scott Kesteven, Ella M M A Martin, et al.
Circulation. Genomic and Precision Medicine|March 21, 2018
A Screening Approach to Identify Clinically Actionable Variants Causing Congenital Heart Disease in Exome DataJustin O Szot, Hartmut Cuny, Gillian M Blue, et al.
Cells|March 11, 2023
Insights into the Role of a Cardiomyopathy-Causing Genetic Variant in ACTN2Sophie Broadway-Stringer, He Jiang, Kirsty Wadmore, et al.
Basic Research in Cardiology|February 27, 2021
Functional analysis of a gene-edited mouse model to gain insights into the disease mechanisms of a titin missense variantHe Jiang, Charlotte Hooper, Matthew Kelly, et al.
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