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Dwi U Kemaladewi

Showing results (1-10 of 17) with videos related to

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Emerging Topics in Life Sciences|February 1, 2021
Development of therapeutic genome engineering in laminin-α2-deficient congenital muscular dystrophyDwi U Kemaladewi, Ronald D Cohn
Trends in Molecular Medicine|February 10, 2016
Exon Snipping in Duchenne Muscular DystrophyDwi U Kemaladewi, Ronald D Cohn
Methods in Enzymology|May 17, 2025
Gene replacement therapy to restore polyamine metabolism in a Snyder-Robinson syndrome mouse modelOluwaseun Akinyele, Krystal B Tran, Marie A Johnson, et al.
Biorxiv : the Preprint Server for Biology|March 31, 2025
Targeting Galectin-3 to modulate inflammation in LAMA2-deficient congenital muscular dystrophyYonne Karoline Tenorio de Menezes, Jinseo Lee, Jia Qi Cheng-Zhang, et al.
Biorxiv : the Preprint Server for Biology|March 22, 2023
CRISPRa-induced upregulation of human <i>LAMA1</i> compensates for <i>LAMA2</i>-deficiency in Merosin-deficient congenital muscular dystrophyAnnie I Arockiaraj, Marie A Johnson, Anushe Munir, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|December 29, 2011
Cell-type specific regulation of myostatin signalingDwi U Kemaladewi, David J J de Gorter, Annemieke Aartsma-Rus, et al.
Biorxiv : the Preprint Server for Biology|January 30, 2023
Impaired polyamine metabolism causes behavioral and neuroanatomical defects in a novel mouse model of Snyder-Robinson SyndromeOluwaseun Akinyele, Anushe Munir, Marie A Johnson, et al.
Disease Models & Mechanisms|March 11, 2024
Impaired polyamine metabolism causes behavioral and neuroanatomical defects in a mouse model of Snyder-Robinson syndromeOluwaseun Akinyele, Anushe Munir, Marie A Johnson, et al.
Plos One|July 18, 2013
Antisense-oligonucleotide mediated exon skipping in activin-receptor-like kinase 2: inhibiting the receptor that is overactive in fibrodysplasia ossificans progressivaSongting Shi, Jie Cai, David J J de Gorter, et al.
Molecular Therapy. Nucleic Acids|April 3, 2014
Targeting TGF-β Signaling by Antisense Oligonucleotide-mediated Knockdown of TGF-β Type I ReceptorDwi U Kemaladewi, Svitlana Pasteuning, Joke W van der Meulen, et al.
Pageof 2

Showing results (1-10 of 17) with videos related to

Sort By:
Pageof 2
Emerging Topics in Life Sciences|February 1, 2021
Development of therapeutic genome engineering in laminin-α2-deficient congenital muscular dystrophyDwi U Kemaladewi, Ronald D Cohn
Trends in Molecular Medicine|February 10, 2016
Exon Snipping in Duchenne Muscular DystrophyDwi U Kemaladewi, Ronald D Cohn
Methods in Enzymology|May 17, 2025
Gene replacement therapy to restore polyamine metabolism in a Snyder-Robinson syndrome mouse modelOluwaseun Akinyele, Krystal B Tran, Marie A Johnson, et al.
Biorxiv : the Preprint Server for Biology|March 31, 2025
Targeting Galectin-3 to modulate inflammation in LAMA2-deficient congenital muscular dystrophyYonne Karoline Tenorio de Menezes, Jinseo Lee, Jia Qi Cheng-Zhang, et al.
Biorxiv : the Preprint Server for Biology|March 22, 2023
CRISPRa-induced upregulation of human <i>LAMA1</i> compensates for <i>LAMA2</i>-deficiency in Merosin-deficient congenital muscular dystrophyAnnie I Arockiaraj, Marie A Johnson, Anushe Munir, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|December 29, 2011
Cell-type specific regulation of myostatin signalingDwi U Kemaladewi, David J J de Gorter, Annemieke Aartsma-Rus, et al.
Biorxiv : the Preprint Server for Biology|January 30, 2023
Impaired polyamine metabolism causes behavioral and neuroanatomical defects in a novel mouse model of Snyder-Robinson SyndromeOluwaseun Akinyele, Anushe Munir, Marie A Johnson, et al.
Disease Models & Mechanisms|March 11, 2024
Impaired polyamine metabolism causes behavioral and neuroanatomical defects in a mouse model of Snyder-Robinson syndromeOluwaseun Akinyele, Anushe Munir, Marie A Johnson, et al.
Plos One|July 18, 2013
Antisense-oligonucleotide mediated exon skipping in activin-receptor-like kinase 2: inhibiting the receptor that is overactive in fibrodysplasia ossificans progressivaSongting Shi, Jie Cai, David J J de Gorter, et al.
Molecular Therapy. Nucleic Acids|April 3, 2014
Targeting TGF-β Signaling by Antisense Oligonucleotide-mediated Knockdown of TGF-β Type I ReceptorDwi U Kemaladewi, Svitlana Pasteuning, Joke W van der Meulen, et al.
Pageof 2